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Renal physiology growth factors

Endothelial cells are the major source of ET-1-synthesis. ET-1 is also produced by astrocytes, neurons, hepatocytes, bronchial epithelial cells, renal epithelial and mesangial cells. Physiological stimuli of ET-1-synthesis in endothelial cells are angiotensin II, catecholamines, thrombin, growth factors, insulin, hypoxia and shear stress. Inhibitors of ET-1 synthesis are atrial natriuretic peptide, prostaglandin E2 and prostacyclin. ET-2 is mainly synthesized in kidney, intestine, myocardium and placenta and ET-3 is predominantely produced by neurons, astrocytes and renal epithelial cells. [Pg.472]

Franklin SC, Moulton M, SIcard GA, Hammerman MR, and Miller SB. Insulln-llke growth factor I preserves renal function postop-eratively.The American Journal of Physiology 272 F257-259,1997. [Pg.83]

Liu Y (2004) Hepatocyte growth factor in kidney fibrosis Therapeutic potential and mechanisms of action. American Journal of Physiology. Renal Physiology 287 F7-F16. [Pg.2585]


See other pages where Renal physiology growth factors is mentioned: [Pg.247]    [Pg.106]    [Pg.42]    [Pg.397]    [Pg.92]    [Pg.285]    [Pg.249]    [Pg.174]    [Pg.641]    [Pg.196]    [Pg.180]    [Pg.95]    [Pg.72]    [Pg.278]    [Pg.2092]   
See also in sourсe #XX -- [ Pg.150 ]




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