Pulmonary emphysema oedema

Ot and photo-oxidants Pulmonary oedema, emphysema, asthma, eye. nose, and throat irritation, reduced lung capacity. Vegetation damage, necrosis of leaves and pines, stunting of growth, photosynthesis inhibitor, probable cause of forest die-back, suspected cause of crop loss. Attack and destruction of natural rubber and polymers, textiles and materials. 

NO, Pulmonary congestion and oedema, emphysema, nasal and eve irritation. Chlorosis and necrosis or leaves. Precursor to acid rain and photo-oxidants. Weathering and corrosion. 

Acute poisoning may result from inhalation of cadmium dusts and fumes (usually cadmium oxide) and from the ingestion of cadmium salts. The major toxic effects are due to local irritation. In the case of oral intake, these include nausea, vomiting, salivation, diarrhoea and abdominal cramp. Cadmium is more toxic when inhaled. Signs and symptoms, which appear after a few hours, include irritation of the upper respiratory tract, chest pains, nausea, dizziness and diarrhoea. Permanent lung damage may occur in the form of emphysema and peribronchial and perivascular fibrosis. Death is usually due to massive pulmonary oedema. 

Focal haemorrhage and pulmonary oedema are typical of the acute toxic effects, but there does not appear to be extensive permanent damage from non-fatal doses. However, residual focal emphysema and interstitial fibrosis remaining after the initial lesions have cleared are possible long term results of the injury [1794,2235]. 

Metal inhalation is the greatest hazard. Within several hours irritation of the airways, nausea, vomiting, chest pain, dizziness, pulmonary oedema, death. Survivors emphysema, nephrotoxicity, osteomalacia. 

Higher amounts of tryptophan (its degradation produd skatole) in feeds of ruminants may cause acute pulmonary oedema (a buildup of fluid in the spaces outside the blood vessels of the lungs) and emphysema (a chronic respiratory disease causing a decrease in lung function and often breathlessness). 

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