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Platelet formation

In addition to the generation of platelets, hydrogenation of silicon also induces electronic deep levels in the band gap. As in the case of platelet formation, these defects are considered to be unrelated to either plasma or radiation damage because they can be introduced with a remote hydrogen plasma. Comparison of depth distributions and annealing kinetics of the platelets and gap states has been used to a limited extent to probe the relationship among these manifestations of H-induced defects. [Pg.146]

Other cytokines synthesised by activated neutrophils (but not bloodstream neutrophils) include interferon-a G-CSF and M-CSF. Interferon-a expression is stimulated by exposure of blood neutrophils to G-CSF (incubation times >3 h are required), but expression of this cytokine is not activated by exposure to either LPS or fMet-Leu-Phe. G-CSF induces 0.95- and 1.2-kb mRNA molecules, the latter transcript containing the message plus a 3 noncoding region. Synthesis of interferon-a protein (as determined by a radioimmunoassay) requires incubation times in excess of 6 h, and the functions of this cytokine include inhibition of neutrophil-colony formation and platelet formation in vitro. [Pg.254]

Busulfan has a selective activity on the bone marrow, suppressing granulocyte and platelet formation, and is primarily used in granulocytic leukaemia. Like the nitrogen mustards it can cause pulmonary fibrosis. [Pg.248]

Nobuoka, S., and Ado, K., Studies on thin iron oxide platelets formation of a-iron oxide by hydrothermal reactions, Shikizai, 60, 265, 1987. [Pg.704]

Fig. 11.12. Schematic illustration of the H-platelet formation and development into H2 gas... Fig. 11.12. Schematic illustration of the H-platelet formation and development into H2 gas...
Myeloma is characterized by bony metastases. and bone p iin is often the presenting symptom. In the face of increasing synthesis of abnonnal itiimuno-globulins, other bone marrow function is reduced, and there is a decline in red and white cell and platelet formation and decreased production of nomial inmiuno-globulins. Anaemia and susceptibility to infection are the consequences. Treatment of myeloma involves the use of bone marrow suppressive dnigs. Plasmapheresis (replacement of the patient s plasma with normal plasma, w ith retention of his or her ow n cellular components) has been used in the treatment of myeloma and macro-globulinaemia. [Pg.112]

Slit flow compatibilization HDPE/PA Modified PA platelet formation, permeability barrier Subramanian, 1985, 1987... [Pg.504]

McKean, M.L., Smith, J.B. and Silver, M.J. (1982). Phospholipid biosynthesis in human platelets. Formation of phosphatidylcholine from 1-acyl-lysophosphatidylcholine by acyl-CoA l-acyl-sn-glycero-3-phosphocholine acyl transferase. ]. Biol Chem., 257, 11278-11283... [Pg.12]

Thromboxanes, TX derivatives of the Prostaglandins (see). T. induce aggregation of platelets, formation of clots and smooth muscle contraction. TXA2 is more active than TXBj, but it decays so rapidly (Fig.) that experimental analysis is difficult. TXB2 does not cause an increase in platelet cAMP levels (in contrast to the prostaglandins), while TXAj inhibits the increase of platelet cAMP caused by prostaglandins. [Pg.671]

Splenic factor inhibits platelet formation in bone marrow by blocking formation of megakaryocytes reticuloendothelial cells platelets... [Pg.412]

Hemostasis in small capillaries is particularly intriguing. It can no longer be explained by capillary constriction or platelet formation. No capillary plugs are observed. [Pg.413]

Figure 17 shows photographs of uncured (a, b) and cured (c, d) 75/25 PAI-2/LCP blends, respectively. M case of uncured 75/25 PAI-2/LCP sauries (Figure 17a, b), the LCP rich skin (a) also shows platelet formation and the PAI-2 rich core (b) shows uneven discontinuous specks of LCP. Interestingly, on curing (Figure 17c, d) the platelets are transformed into fibrils in the skin (c) and the LCP ecks into fibrils in the core (d). No orientation of the fibrils is observed. In both uncured and cured sanq>les, phase separation is evident in the core. [Pg.161]

The Equation (14) and Equation (18) describe theoretically the experimental dependences of nanocomposites polymer/organoclay combustibility on nanofiller stmc-ture. So, organoclay contents cp increasing results in X reduction and, respectively, in Q decrease. The transition from organoclay exfoliated stracture up to intercalated one results in a packet (tactoid) from N organoclay platelets formation, that increases... [Pg.235]

Review articles have appeared which discuss the effects of PG s on blood pressure, " platelet formation, cell-membrane penetration by calciiun... [Pg.307]


See other pages where Platelet formation is mentioned: [Pg.136]    [Pg.38]    [Pg.143]    [Pg.146]    [Pg.353]    [Pg.67]    [Pg.263]    [Pg.131]    [Pg.72]    [Pg.23]    [Pg.128]    [Pg.131]    [Pg.96]    [Pg.534]    [Pg.2859]    [Pg.534]    [Pg.198]    [Pg.107]    [Pg.296]    [Pg.590]    [Pg.451]    [Pg.316]    [Pg.28]    [Pg.170]    [Pg.154]    [Pg.69]    [Pg.216]    [Pg.46]   
See also in sourсe #XX -- [ Pg.152 ]

See also in sourсe #XX -- [ Pg.43 ]




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