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Nucleus raphe magnus

B8 Caudal linear nucleus B3 Nucleus Raphe magnus... [Pg.189]

Figure 22.8 The distribution of brainstem Raphe nuclei. Neurons that release 5-HT are clustered in two groups of nuclei in the pons and upper brainstem. The superior group, which projects to forebrain areas, includes the dorsal Raphe nucleus (DRN) and the median Raphe nucleus (MRN). The inferior group projects to the medulla and spinal cord and includes the nucleus Raphe pallidus (NRP), the nucleus Raphe obscurus (NRO) and the nucleus Raphe magnus (NRM)... Figure 22.8 The distribution of brainstem Raphe nuclei. Neurons that release 5-HT are clustered in two groups of nuclei in the pons and upper brainstem. The superior group, which projects to forebrain areas, includes the dorsal Raphe nucleus (DRN) and the median Raphe nucleus (MRN). The inferior group projects to the medulla and spinal cord and includes the nucleus Raphe pallidus (NRP), the nucleus Raphe obscurus (NRO) and the nucleus Raphe magnus (NRM)...
Figure 8.2 The endogenous analgesic system. The three major components of the endogenous analgesic system include the periaqueductal gray matter in the midbrain nucleus raphe magnus in the medulla and pain inhibitory complex in the dorsal horns of the spinal cord. This system causes presynaptic inhibition of pain fibers entering the spinal cord. The binding of enkephalin to opioid receptors on the pain fibers prevents release of the neurotransmitter, substance P. As a result, the pain signal is terminated in the spinal cord and does not ascend to higher centers in the CNS. Figure 8.2 The endogenous analgesic system. The three major components of the endogenous analgesic system include the periaqueductal gray matter in the midbrain nucleus raphe magnus in the medulla and pain inhibitory complex in the dorsal horns of the spinal cord. This system causes presynaptic inhibition of pain fibers entering the spinal cord. The binding of enkephalin to opioid receptors on the pain fibers prevents release of the neurotransmitter, substance P. As a result, the pain signal is terminated in the spinal cord and does not ascend to higher centers in the CNS.
Iwamoto FT. (1991). Characterization of the antinociception induced by nicotine in the pedunculopontine tegmental nucleus and the nucleus raphe magnus. J Pharmacol Exp Ther. 257 120-33. [Pg.524]

Behbehani, M. M. and Fields, H. L. Evidence that an excitatory connection between the periaqueductal grey and nucleus raphe magnus mediated stimulation produced analgesia, Brain Res. 1979, 170, 85-93. [Pg.280]

Bowker, R. M. and Dilts, R. P. Distribution of mu-opioid receptors in the nucleus raphe magnus and nucleus gigantocellularis a quantitative autoradiographic study, Neurosci. Lett. 1988, 88, 247-252. [Pg.280]

Giesler, G. J., Gerhart, K. D., Yezierski, R. P., Wilcox, T. K., Willis, W. D. Postsynaptic inhibition of primate spinothalamic neurons by stimulation in nucleus raphe magnus, Brain Res. 1981, 204, 184-188. [Pg.281]

Bitner, R.S., Nikkei, A.L., Curzon, P., Arneric, S.P., Bannon, A.W., Decker, M.W., 1998. Role of the nucleus raphe magnus in antinociception produced by ABT-594 immediate early gene responses possibly linked to neuronal nicotinic acetylcholine receptors on serotonergic neurons. J. Neurosci. 18, 5426-5432. [Pg.53]

Richardson JD, KUo S, Hargreaves KM (1998b) Cannabinoids reduce hyperalgesia and inflammation via interaction with peripheral CBj receptors. Pain 75 111-119 Schroeder K, Fahey T (2002) Systematic review of randomised controlled trials of over the counter cough medicines for acute cough in adults, Br Med J 324 1-6 Sessle BJ, Ball GJ, Lucier GE (1981) Suppressive influences from periaqueductal gray and nucleus raphe magnus on respiration and related reflex activities and on solitary tract neurons, and effect of naloxone. Brain Res 216(1) 145-161... [Pg.76]

FIGURE 40.6 Neurons Fos positive in the (a) motor cortex, (b) locus ceruleus, (c) nucleus raphe magnus, and (d) periaqueductal grey. Vehicle (control) or EO-pCD (6,12 and 24 mg/kg) were administered orally 1.5 h before perfusion. Values represent mean SEM (n = 6 per group). p < 0.05, p < 0.01 or p < 0.001 versus control (one-way ANOVA followed by Tukey s test). (From Siqueira-Lima, P.S. et al.. Bask Clin. Pharmacol. Toxicol., 114(2), 188, 2014.)... [Pg.882]

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See also in sourсe #XX -- [ Pg.83 ]

See also in sourсe #XX -- [ Pg.57 ]



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Raphe nuclei

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