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Neurotoxicity of mercury

Chang YC, Yeh C, Wang JD. 1995. Subclinical neurotoxicity of mercury vapor revealed by a multimodality evoked potential study of chloralkali workers. Amer J Ind Med 27(2) 271-279. [Pg.591]

Marino PE, Landrigan PJ, Graef J, et al A case report of lead paint poisoning during renovation of a Victorian farmhouse. Am J Public Health 80 1183-1185, 1990 Markowitz SB, Nunez CM, Klitzman S, et al Lead poisoning due to Hai Ge Fen the porphyrin content of individual erythrocytes. JAMA 271 932-934, 1994 Marsh DO The neurotoxicity of mercury and lead, in Neurotoxicity of Industrial and Commercial Chemicals, Vol 1. Edited by O Donoghue JL. Boca Raton, FL, CRC Press, 1985, pp 159-169... [Pg.133]

As mentioned in Sect. C.II.2, above, inhaled mercury vapor persists in the blood stream sufficiently long to reach the blood-brain barrier. Due to its high lipophilicity, mercury vapor rapidly diffuses across the blood-brain barrier, where it is oxidized to ionic mercury by the catalase-compound I system. Oxidized mercury is easily subjected to the reaction with SH-containing ligand in the central nervous system thus fixation of mercury occurs. Little is known concerning the precise causal relationship between the sites of mercury deposition in the brain and the characteristic neurotoxicity of mercury vapor. It is conceivable that essentially the same mechanism of toxicity as that of ionic mercury is operating at molecular level. [Pg.179]

See other pages where Neurotoxicity of mercury is mentioned: [Pg.1622]   
See also in sourсe #XX -- [ Pg.2 , Pg.21 , Pg.26 , Pg.27 , Pg.28 , Pg.29 , Pg.30 , Pg.31 , Pg.32 , Pg.33 , Pg.34 , Pg.35 , Pg.36 , Pg.37 , Pg.38 , Pg.39 , Pg.40 , Pg.41 ]



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