Leads augmented

Concrete is commonly used, augmented by iron and lead for gamma rays and water for fast neutrons. 

The augmentation of the work of adhesion by acid-base effects has, as a consequence, an effect on wettability properties. Identifying /sl in Eq. 26 as W and substituting it into Eq. 3 and Eq. 14, respectively leads to ... 

Higher melt temperatures lead to an increase in band intensity and merging thereof, as shown in Fig. 84. The merging of bands that occurs at increased temperatures can be explained by the augmentation of ion diffusion that causes an averaging of the potential of inter-ionic interactions between the NbF6 ions and the outer-sphere cations. 

Activation of Mi, M3, and M5 mAChRs does not only lead to the generation of IP3 followed by the mobilization of intracellular Ca2+, but also results in the stimulation of phospholipase A2, phospholipase D, and various tyrosine kinases. Similarly, M2 and M4 receptor activation does not only mediate the inhibition of adenylyl cyclase, but also induces other biochemical responses including augmentation of phospholipase A2 activity. Moreover, the stimulation of different mAChR subtypes is also linked to the activation of different classes of mitogen-activated protein kinases (MAP kinases), resulting in specific effects on gene expression and cell growth or differentiation. 

The natural fluorescence of ergot alkaloids is considerably augmented by the reagent [1]. Heating for longer than 3 min or to more than 80 °C leads to a reduction in fluorescence intensity and, hence, should be avoided [3]. 

The ability of the E-plastomers to participate in the peroxide-mediated chain-extension processes can be augmented in blends with EPDM, where the mixture is homogeneously cross-linked with free radicals [5]. The use of these blends of EPDM and E-plastomers leads to improved processing and physical properties of the combination, compared to the EPDM alone, though the resulting vulcanizates are somewhat harder than the EPDM vulcanizates alone due to the presence of the semicrystalline plastomers in the vulcanized mixture. 

Amylase enters the blood largely via the lymphatics. An increase in hydrostatic pressure in the pancreatic ducts leads to a fairly prompt rise in the amylase concentration of the blood. Neither an increase in volume flow of pancreatic juice nor stimulation of pancreatic enzyme production will cause an increase in senm enzyme concentration. Elevation of intraductal pressure is the important determinant. Stimulation of flow in the face of obstruction can, however, augment the entry of amylase into the blood, as can disruption of acinar cells and ducts. A functional pancreas must be present for the serum amylase to rise. Serum amylase determination is indicated in acute pancreatitis in patients with acute abdominal pain where the clinical findings are not typical of other diseases such as appendicitis, cholecystitis, peptic ulcer, vascular disease or intestinal obstruction. In acute pancreatitis, the serum amylase starts to rise within a few hours simultaneously with the onset of symptoms and remains elevated for 2 to 3 days after which it returns to normal. The peak level is reached within 24 hours. Absence of increase in serum amylase in first 24 hours after the onset of symptoms is evidence against a diagnosis of acute pancreatitis (76). 

Figure 17.7 Possible mechanism by which atypical neuroleptics with antimuscarinic activity produce few EPSs. Normally the inhibitory effects of DA released from nigrostriatal afferents on to striatal neuron D2 receptors is believed to balance the excitatory effect of ACh from intrinsic neurons acting on muscarinic (M2) receptors (a). Typical neuroleptics block the inhibitory effect of DA which leaves unopposed the excitatory effect of ACh (b) leading to the augmented activity of the striatal neurons and EPSs (see Fig. 15.2). An atypical neuroleptic with intrinsic antimuscarinic activity reduces this possibility by counteracting the excitatory effects of released ACh as well as the inhibitory effects of DA (c). Thus the control of striatal neurons remains balanced...

In this section we derive a system equation which describes a drifting calibration line. Let us suppose that the intercept jC (/ + 1) at a time y + 1 is equal to xfj) at a time j augmented by a value a(j), which is the drift. By adding a non-zero system noise to the drift, we express the fact that the drift itself is also time dependent. This leads to the following equations [5,6] ... 

Animal studies have shown that the periodic dosing regimen often leads to behavioral augmentation or sensitization in animals (Post 1981). 

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