Latency-limited applications

In order to match the required throughput or the required array size, it is necessary to partition the initial architecture with full index ranges into a set of serially executed subsets and then to cluster these onto a smaller sized array. This important task has been addressed in depth [5] and is described in chapter 4. Extensions which can result in a better preservation of the regularity, especially for latency-limited applications, are proposed in chapter 3. 

In latency-limited applications, the important criterion is the execution time T. This is in contrast to the real-time signal and data processing applications, where the period P is more important. Solutions for the latter category are the focus of chapters 5 and 6. In this chapter, the latency-limited case will be addressed. Even though a large part of the underlying theory can be shared, important distinctions will be pointed out. 

Both linear and piece-wise linear mappings can be derived in a systematic way, and methods exist to optimize given criteria within a constraint on execution time (for latency-limited applications), the period (for throughput-limited processing), or the number of processors (for area-bound applications). 

Most of the data located concerning the health effects of heptachlor and heptachlor epoxide in humans come from case reports and occupational epidemiology studies of workers engaged either in the manufacture or application of pesticides. There is some information on people who have consumed heptachlor-contaminated food or dairy products, but no adverse health effects have been related to these exposures. The occupational studies involve exposures that are predominantly inhalation with contributions from dermal exposure, whereas all the animal studies were conducted using oral or intraperitoneal exposures. The occupational and case reports provide no quantitation of dose or duration of exposure, which makes it impossible to determine with any precision the effect levels for humans. There are no data that indicate that heptachlor or heptachlor epoxide are carcinogenic to humans. However, human studies are limited by the long latency period of carcinogenesis and by ascertainment and follow-up biases. 

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