Hypoglycemia insulin

The response of prolactin (PRL) to the hypothalamic TSH-releasing hormone (TRH) and to insulin hypoglycemia is reduced in long-term Li+-treated patients, as is the response of growth hormone to insulin hypoglycemia however the concentration of growth hormone is also reported to be elevated during Li+ treatment [170]. 

Other tests include the insulin hypoglycemia test, the metyrapone test, and the corticotrophin-releasing hormone stimulation test. 

Human insulin Hypoglycemia and hypokalemia are among the potential clinical adverse reactions associated with the use of all insulins. Other adverse reactions... 

In 18 subjects with type 1 diabetes, mean age 37 years, who received in random order on separate days pramlintide 60 micrograms or placebo plus their usual doses of regular insulin, hypoglycemia (pramlintide 28% versus 16%) and mild nausea (17% versus 11%) were the most... 

Autoimmune insulin hypoglycemia is rare, and is most often seen in East Asia. Patients have low concentrations of insulin and C-peptides, but insulin can be released from... 

Anesthesia alone and the stimulation of the hypothalamopituitary adrenal axis by insulin hypoglycemia or a pyrogen response failed to alter the concentration of free thyroxine. 

Despite the apparent importance of the adrenal medulla in homeostasis, particularly regulation of metabolism, the medulla in contrast to the adrenal cortex is not vital for survival. Studies in adrenalectomized subjects clearly show that both hemodynamic and glucose-counter-regulatory responses to insulin-hypoglycemia, exercise, and other manipulations remain intact despite absence of epinephrine responses. This contrasts with the severe disturbances of blood pressure regulation accompanying loss of sympathetic nerves. 

Schmitz, O., and Moller, J., Impaired prolactin response to arginine infusion and insulin hypoglycemia in chronic renal failure. Acta Endocrinol. (Copenhagen) 102, 486-491 (1983). 

Hypoglycemia is a common side effect of treatment with insulin, and, in insulin-treated diabetics admitted with severe hypoglycemic attacks, pronounced abnormalities in plasma ALT and AST activities have been reported (G5, S31). In contrast, when prospective studies using controlled insulin hypoglycemia have been performed few abnormalities in the activities of AST and ALT have been found when there was no evidence of preexisting liver disease nevertheless, abnormalities in liver function tests are common in diabetic patients (S2, P3). 

Corticotropin secretion is unaff ed by GH-RIH in normal subjects (H3) both basally and in response to insulin hypoglycemia. However, Tyrrell et al. (Tl) have shown a significant faJl in elevated ACTH levels in Nelsons syndrome (4 patients) and in Cushing s disease (1 patient) in response to GH-RIH infusions. These results suggest that the mechanism determining release of ACTH by adenoma cells differ from that in normal pituitary cells. 

Feise G, Kogure K, Busto R, Scheinberg P, Reinmuth O. Effect of insulin hypoglycemia upon cerebral energy metabolism and EEG activity in the rat. Brain Res 1976. 126 263-280 Harris Rl, Wieloch T, Symon L, Siesjo BK. Cerebral extracellular calcium activity in severe hypoglycemia Relation to extracellular potassium and energy state. J Cereb Blood Elow Metab 1984. 4 187-193... 

Butterworth R. F., Merkel A. D., and Landreville F. (1982) Regional ammo acid distribution m relation to function m insulin hypoglycemia. J Neurochem, 38, 1483-1489. 

It is believed that insulin hypoglycemia brings about gastrin secretion by stimulation of the long vagal reflexes. Distension of the stomach is likely to release... 

Code himself laboriously determined the histamine content of the gastric mucosa in many secretory states, but he could find no significant change except after insulin hypoglycemia.If, as Georg Kahlson believed, the histamine content of the gastric mucosa is the result of rates of histamine formation and histamine release, measurement of the histamine content at any one time provides little evidence about histamine as the final common mediator. 

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