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Insulin resistance glucose phosphorylation

With respect to glycaemic parameters FXR-deficient mice (FXR ) show impaired glucose tolerance, whole-body glucose disposal and insulin resistance. FXR deficiency is also associated with impaired distal insulin signalling in muscle and WAT through reduced insulin-dependent AKT phosphorylation. ... [Pg.133]

After insulin binds to insulin receptor on cell surface, insulin receptor and its substrates are phosphorylated, which leads to activation of various insulin signaling pathways [202]. Reynoso et al. [203] evaluated several aspects of the insulin resistance induced by palmitic acid in rats and found that after treatment with 0.09 g/kg of palmitic acid there is a delay in the curve of tolerance to glucose. The authors concluded that occur an increase in the phosphorylations in serine of the insulin receptor after the treatment with palmitate, suggesting that PKC has a role as negative regulator of the insulin receptors activation in the insulin resistance induced by palmitic acid. [Pg.92]


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See also in sourсe #XX -- [ Pg.84 ]




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