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Inositol metabolism

HMIT is a H+-coupled myo-inositol symporter. High levels of its expression are observed in neurons and glia of hippocampus, hypothalamus, cerebellum and brainstem. Since myo-inositol is a precursor for phosphatidyl inositol, which itself is a critical regulator of many neuronal processes (Ch. 20), HMIT regulation is possibly involved in various mood and behavior patterns that are affected by inositol metabolism and by pharmacologic agents that modify inositol metabolism (see Chs 54 and 55). [Pg.91]

Cecil KM, Patel NC, DelBello MP (2006) Inositol metabolism in pediatric bipolar disorders. Int J ain Neuropsychol 3 177-183... [Pg.139]

Johnson RD, Minneman KP (Xj-Adrenergic receptors and stimulation of [3H]inositol metabolism in rat brain regional distribution and parallel inactivation. Brain Res 341 7-15, 1985... [Pg.667]

A selection of other tumor suppressor genes is summarized in Table 14.2. Interestingly, an enzyme of phosphatidyl-inositol metabolism has been also identified as a tumor suppressor. The PTEN tumor suppressor gene codes for a phospholipid phosphatase which specifically cleaves a phosphate from the second messenger phosphatidyl-inosi-tol-3,4,5-trisphosphate (PtdInsPj, see 6.6.2). and thus inactivates the messenger (review Maehama and Dixon, 1999). ... [Pg.452]

The myo-isomer is the most abundant form in nature. It occupies a unique place in inositol metabolism because this is the only isomer synthesized de novo from D-glucose-6-phosphate all other isomers are derived from myo-inositol (Loewus, 1990b Loewus and Murthy, 2000). In early 1900s, myo-inositol was called meso-inositol (abbreviated ms-inositol) probably because, like mesotar-taric acid, it does not exhibit optical activity and cannot be resolved into optical isomers (Posternak, 1965). However, as discussed above, six other isomers also do not exhibit optical activity so the name seemed inappropriate. In 1954, the name myo-inositol was introduced - a name not particularly well coined either since it is a pleonasm - both myo and inositol mean muscle. Nevertheless, the name persists. [Pg.5]

Loewus, F.A., 1990a, Inositol biosynthesis, metabolism Precursor role and breakdown. In Morre, D.J., Boss, W.F., and Loewus, FA. (eds.), Inositol Metabolism in Plants. Wiley-Liss, New York, NY, pp. 13-19. [Pg.20]

Comparative study of 3H-labeled myo- and vty//u-inositol metabolism in maturing wheat... [Pg.32]

Chen, M., and Loewus, F.A., 1977, myo-Inositol metabolism in Lilium longiflorum pollen. Uptake and incorporation of myo-inositol-2-3H. Plant Physiol. 59 653-657. [Pg.38]

Flores, S., and Smart, C.C., 2000, Abscisis acid-induced changes in inositol metabolism in... [Pg.39]

Kroh, M., Miki-Hirosige, H., Rosen, W., and Loewus, F., 1970a, Inositol metabolism in plants. VII. Distribution and utilization of label from myo-inositol-U-14C and -2-3H by detached flowers and pistils of Lilium longiflorum. Plant Physiol. 45 86-91. [Pg.41]

Loewus, F.A., and Kelly, S., 1963, Inositol metabolism in plants, I. Labeling patterns in cell wall polysaccharides from detached plants given myo-inositol-2-t or -2-14C. Arch. Biochem. Biophys. 102 96-105. [Pg.42]

Roberts, R.M., Deshusses, J., and Loewus, F., 1968, Inositol metabolism in plants. V Conversion of myo-inositol to uronic acid and pentose units of acidic polysaccharides in root tips of Zea mays. Plant Physiol. 43 979-989. [Pg.44]

Verma, D.C., Tarvares, I, and Loewus, F.A., 1976, Effect of benzyladenine, 2,4-dichlorophenoxyacetic acid, and D-glucose in myo-inositol metabolism in Acer pseudoplatanus L. cells grown in suspension culture. Plant Physiol. 37 241-244. [Pg.46]

Loewus, F, 1965, Inositol metabolism and cell wall formation in plants. Fed. Proc. 24 855-862. [Pg.66]

Nelson, D.E., Rammesmayer, G., and Bohnert, H.J., 1998, Regulation of cell-specific inositol metabolism and transport in plant salinity tolerance. Plant Cell 10 753-764. [Pg.68]

Acharya, J.K., Labarca, P., Delgado, R., Jalink, K., and Zuker, C.S., 1998, Synaptic defects and compensatory regulation of inositol metabolism in inositol polyphosphate 1-phosphatase mutants. Neuron 20 1219-1229. [Pg.96]

Kagiwada, S., Hosaka, K., Murata, M., Nikawa, J., and Takatsuki, A., 1998, The Saccharomyces cerevisiae SCS2 gene product, a homolog of a synaptobrevin-associated protein, is an integral membrane protein of the endoplasmic reticulum and is required for inositol metabolism. J. Bacteriol. 180 1700-1708. [Pg.152]

See other pages where Inositol metabolism is mentioned: [Pg.448]    [Pg.116]    [Pg.41]    [Pg.41]    [Pg.42]    [Pg.42]    [Pg.47]    [Pg.49]    [Pg.51]    [Pg.52]    [Pg.53]    [Pg.55]    [Pg.57]    [Pg.59]    [Pg.59]    [Pg.61]    [Pg.63]    [Pg.63]    [Pg.65]    [Pg.67]    [Pg.67]    [Pg.69]    [Pg.179]    [Pg.179]   
See also in sourсe #XX -- [ Pg.7 , Pg.8 , Pg.9 , Pg.10 , Pg.11 , Pg.45 ]

See also in sourсe #XX -- [ Pg.720 ]



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