Hyperventilation chest pain

Bumetanide Adverse reactions may include impaired hearing, ear discomfort, dry mouth, pain, renal failure, weakness, arthritic pain, muscle cramps, ECG changes, chest pain, hives, pruritus, itching, sweating, hyperventilation. 

Respiratory Pneumonia pharyngitis sinusitis hyperventilation rhinitis apnea aspiration pneumonia asthma dyspnea atelectasis increased cough/sputum epistaxis hypoxia pneumothorax hemoptysis bronchitis chest pain pulmonary fibrosis. 

Disuifiram-aicohoi reaction Disulfiram plus alcohol, even small amounts, produces flushing, throbbing in head and neck, throbbing headaches, respiratory difficulty, nausea, copious vomiting, sweating, thirst, chest pain, palpitations, dyspnea, hyperventilation, tachycardia, hypotension, syncope, marked uneasiness, weakness, vertigo, blurred vision, and confusion. In severe reactions there may be respiratory depression, cardiovascular collapse, arrhythmias. Ml, acute CHF, unconsciousness, convulsions, and death. The intensity of the reaction is proportional to the amounts of disulfiram and alcohol ingested. The duration of the reaction varies from 30 to 60 minutes to several hours. 

Anxiety symptoms are an inherent part of the initial clinical presentation of several diseases, thus complicating the distinction between anxiety disorders and medical disorders. If the anxiety symptoms are secondary to a medical illness, they usually will subside as the medical situation stabilizes. However, the knowledge that one has a physical illness (e.g., cancer and diabetes) can trigger anxious feelings and further complicate therapy. Persistent anxiety subsequent to a physical illness requires further assessment for an anxiety disorder. Symptoms of anxiety frequently present in medical disorders include palpitations, tachycardia, chest pain or tightness, shortness of breath, and hyperventilation. Medical disorders most closely associated with anxiety are listed in Table 69-1. " About 50% of patients with GAD have irritable bowel syndrome. ... 

The majority of patients with pulmonary hypertension are largely asymptomatic until marked vascular alterations have developed. When blood flow through the pulmonary artery is obstructed over a long period of time, however, the clinical picture is predictable and markedly uniform. In general, the patients exhibit normal pulmonary function measurements, a low carbon monoxide diffusion capacity (DlCO), and marked hyperventilation that leads to hypocapnia and decreased serum bicarbonate concentrations. Additional symptoms include weakness, fatigue, exertional dyspnea, and chest pains upon exertion due to low cardiac output and hypoxemia. Occasionally, hoarseness, hemoptysis, and cyanosis occur. 

Autonomic hyperactivity (heart pounding, sweating, chest pains), hyperventilation... 

A review of symptoms will enable the medical officer to evaluate the ability of an individual to work in protective ensemble. Questions concerning shortness of breath or labored breathing on exertion, asthma, other respiratory symptoms, chest pain, high blood pressure, and heat intolerance will provide helpful information. Questions about allergic reactions to rubber products and cold-induced bronchospasm should be asked and a brief psychiatric history directed toward the individual s ability to be encapsulated in personal protective equipment should be taken. Questions about panic attacks, syncopal episodes, or hyperventilation will also offer valuable information. 

A 42-year-old woman with neck pain took 15 g of maqianzi in two doses 7 hours apart (recommended dose 0.3-0.6 g) (2). One hour after she took the second dose she suddenly developed tonic contractions of all her limbs and carpopedal spasm lasting 5 minutes, difficulty in breathing, chest discomfort, and perioral numbness. She complained of muscle pain and tiredness and had hyperventilation and weakness of all four limbs. All her symptoms gradually subsided over the next few hours. 

Respiratory alkalosis is associated with many illnesses. Hyperventilation has several causes. The CNS respiratory centre is stimulated by many factors including anxiety, psychosis, pain and fever. Overdosage of salicylates can initially stimulate ventilation causing respiratory alkalosis which may be followed by metabolic acidosis. Stimulation of the chest receptors by conditions such as pneumothorax, pulmonary embolism and pulmonary oedema can cause hyperventilation and hypocapnia. Other causes include mechanical ventilation, hepatic failure and sepsis... 

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