Hexacosanoic acid

Hexachlorocyclotriphosphazene, 19 55 Hexachloroethane, 6 269 Hexachloromelamine, 23 111 Hexachlorophene, bioremediation substrate, 3 773-776 Hexachlorotitanates, 25 53 Hexacosanoic acid, physical properties, 5 30t... 

In addition to their mandibles, which are used to bite, pierce, shear, and cut intruders, soldiers of Coptotermes formosanus, a Formosan subterranean termite, are also armed with chemical weaponry. When disturbed, the soldiers secrete a gluelike white fluid from the frontal gland, which often form a drop between the open mandibles. Lignoceric and hexacosanoic acids were the two major components identified in this secretion [183]. 

Figure 25. -T diagram for racemic methyl ester of 2-methyl-hexacosanoic acid. Derived from Figure 21. Form Lundquist (78). Permission of Stockholm University Library. 

A second important difference between mitochondrial and peroxisomal fi oxidation in mammals is in the specificity for fatty acyl-CoAs the peroxisomal system is much more active on very-long-chain fatty acids such as hexacosanoic acid (26 0) and on branched-chain fatty acids such as phytanic acid and pristanic acid (see Fig. 17-17). These less-common fatty acids are obtained in the diet from dairy products, the fat of ruminant animals, meat, and fish. Their catabolism in the peroxisome involves several auxiliary enzymes unique to this organelle. The inability to oxidize these compounds is responsible for several serious human diseases. Individuals with Zellweger syndrome are unable to make peroxisomes and therefore lack all the metabolism unique to that organelle. In X-linked adrenoleukodystrophy (XALD), peroxisomes fail to... 

O C o fatty acids AWAWC hexadecanoic acid (cutin) hexacosanoic acid (wax)... 

C26H5202 hexacosanoic acid 506-46-7 100.00 0,8198 1 34514 C27H4405 spirostan-2,3,15-triol, (2alpha,3beta,5alpha 511-34-2 25.00 1.0138 2... 

C25H51CXDOH 1-hexacosanoic acid cerotic acid C25COOH... 

Three genetic disorders Zellweger s syndrome, neonatal adrenoleukodystrophy, and childhood adrenoleukodys-trophy) exhibit defective formation of peroxisomes (in Zellweger s syndrome no morphologically detectable peroxisomes are present) or deficiency of one or more constituent enzymes. All three disorders are characterized by a marked accumulation of very long chain, saturated, unbranched fatty acids (tetracosanoic and hexacosanoic acids) in liver and central nervous system tissues, severe neurological symptoms, and early death. 

A variety of biochemical defects have been reported in patients with the Zellweger syndrome. These include a defect in the catabolism of pipecolic acid (D2) and increased plasma, biliary, and urinary levels of intermediates in bile acid synthesis (H4, M15, M30). More recently, the accumulation of very-long-chain fatty acids, such as hexacosanoic acid, has been noted (M32) and elevated plasma phytanic acid with decreased fibroblast phytanic acid oxidase activity reported (P13). 

Hexacosanoic acid, ester with oiean-12-en-3-ol, (3P)- 3-amyrenyl hexacosanoate ... 

Heptenoic acid H2C=CH-(CH2)4-C00H 6-Heptenoic acid, 5-methyl-H2C=CH-CH(CH3)-(CH2)3-C00H Hexacosanoic acid cerotinic acid ... 

Hexacosanoic acid, 24-methyl-Hexadecadienoic acid palmitolenic acid ... 

---