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Hepatocyte growth factor, neutrophil

Exposure of the SECs to pathogens or cytokines produced by other cells during stress induces activation of the SECs and subsequent production of cytokines, eicosanoids, and/or adhesion molecules. For instance, after activation with EPS, a main component of the walls of gramnegative bacteria and a major inducer of inflammation and non-specific immune functions [20], SECs produce a number of pro- and anti-inflammatory cytokines. Pro-inflammatory cytokines shown to be produced were tumour necrosis factor alpha (TNFa) [26] interleukin-1 alpha/beta(IL-lo/p) [27] the major inducer of acute phase proteins interleukin-6 (IL-6) [28] and the neutrophil chemo-attractant interleukin-8 (IL-8) [29]. Anti-inflammatory cytokines shown to be produced were interleukin-10 (IL-10) [27] and hepatocyte growth factor (HGF) [30]. [Pg.93]

McCourt M, Wang JH, Sookhai S, Redmond HP Activated human neutrophils release hepatocyte growth factor/scatter factor. Eur J Surg Oncol 2001 27 396 03. [Pg.179]


See other pages where Hepatocyte growth factor, neutrophil is mentioned: [Pg.2584]    [Pg.185]    [Pg.246]    [Pg.247]    [Pg.54]    [Pg.169]    [Pg.106]    [Pg.220]   


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Neutrophils

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