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Escherichia coli antibiotic resistance

A study with a strain of plasmid-bome antibiotic-resistant Escherichia coli indicated that the strain did not transmit these plasmids to indigenous strains after introduction into the terrestrial environment (Devanas et al. 1986). [Pg.226]

Complicated exacerbation FEV, less than 50% predicted Comorbid cardiac disease Greater than or equal to 3 exacerbations per year Antibiotic therapy in the previous 3 months Above organisms plus drug-resistant pneumococci, P-lactamase-producing H. influenzae and M. catarrhalis, Escherichia coli, Proteus spp., Enterobacter spp., Klebsiella pneumoniae Oral P-Lactam/P-Iactamase inhibitor (amoxicil 1 i n-clavulanate) Fluoroquinolone with enhanced pneumococcal activity (levofloxacin, gemifloxacin, moxifloxacin) Intravenous P-Iactam/P-Iactamase inhibitor (ampicillin-sulbactam) Second- or third-generation cephalosporin (cefuroxime, ceftriaxone) Fluoroquinolone with enhanced pneumococcal activity (levofloxacin, moxifloxacin)... [Pg.241]

Empiric antibiotic therapy is an appropriate approach to traveler s diarrhea. Eradication of the causal microbe depends on the etiologic agent and its antibiotic sensitivity. Most cases of traveler s diarrhea and other community-acquired infections result from enterotoxigenic (ETEC) or enteropathogenic (EPEC) Escherichia coli. Routine stool cultures do not identify these strains primary empiric antibiotic choices include fluoroquinolones such as ciprofloxacin or levofloxacin. Azithromycin may be a feasible option when fluoroquinolone resistance is encountered. [Pg.315]

Perfetto EM, Gondek EK. Escherichia coli resistance in uncomplicated urinary tract infection a model for determining when to change first-line empirical antibiotic choice. Manag Care Interface 2002 6 35M2. [Pg.1158]

The preceding protocol can be successfully applied, essentially without modifications, to prepare active cell-free extracts from bacteria other than Escherichia coli (e.g., Bacillus stearothermophilus and clinical isolates of Pseudomonas aeruginosa bearing multiple antibiotic resistance). [Pg.270]

Watkinson AJ, Micalizzi GB, Graham GM et al (2007) Antibiotic-resistant Escherichia coli in waste waters, surface waters, and oysters from an urban riverine system. Appl Environ Microbiol 73(17) 5667-5670... [Pg.206]

Watkinson AJ, Micalizzi GR, Bates JR et al (2007) Novel method for rapid assessment of antibiotic resistance in Escherichia coli isolates from enviroirmental waters by use of a modified chromogenic agar. Appl Environ Microbiol 73(7) 2224—2229... [Pg.207]

Boczek LA, Rice EW, Johnston B et al (2007) Occurrence of antibiotic-resistant uropathogenic Escherichia coli clonal group A in waste water effluents. Appl Environ Microbiol 73(13) 4180-4184... [Pg.208]

Laroche E, Pawlak B, Berthe T et al (2009) Occurrence of antibiotic resistance and class 1,2 and 3 integrons in Escherichia coli isolated from a densely populated estuary (Seine, France). FEMS Microbiol Ecol 68(1) 118-130... [Pg.211]

Chen B, Zheng W, Yu Y et al (2011) Class 1 integrons, selected virulence genes, and antibiotic resistance in Escherichia coli isolates from the Minjiang River, Fujian Province, China. Appl Environ Microbiol 77(1) 148-155... [Pg.211]

Most infections are caused by gram-negative bacteria, mostly Escherichia coli. In recurrent UTI, after repeated courses of antimicrobial therapy, other organisms and antibiotic resistance can be expected. [Pg.528]

Schrag S.J., V. Perrot, and B.R. Levin (1997). Adaptation to fitness costs of antibiotic resistance in Escherichia coli. Proceedings of the Royal Society of London Series B— Biological Sciences 264 1287-1291. [Pg.283]

S ATP + hygromycin B <1> (<1> responsible for resistance against hygromycin B, an aminocyclitol antibiotic [1] resistance to hygromycin B also occurs in Escherichia coli, where a plasmid encodes a hygromycin B phos-... [Pg.56]

O Hara, K. Kanda,T. Kono, M. Structure of a phosphorylated derivative of oleandomycin, obtained by reaction of oleandomycin with an extract of an erythromycin-resistant strain of Escherichia coli. J. Antibiot., 41,823-827 (1988)... [Pg.169]

H. Umezawa, S. Umezawa, T. Tsuchiya, and Y. Okazaki, 3, 4 -Dideoxy kanamycin B active against kanamycin-resistant Escherichia coli and Pseudomonas aeruginosa, J. Antibiot. 24 485 (1971). [Pg.150]

Xu M, Zhou YN, Goldstein BP et al (2005) Cross-resistance of Escherichia coli RNA polymerases conferring rifampin resistance to different antibiotics. J Bacteriol 187 2783-2792... [Pg.146]

Multicopy plasmids offer a limited, but still instructive comparison. Plasmids are replicating clusters of genes in bacteria. Under certain conditions they confer beneficial traits (such as resistance against antibiotics) on the bacterial host. They can also be passed on by conjugation between cells. A well elucidated mechanism of copy number control of abundant plasmids relies on a frans-acting inhibitor and czs-acting activator. For example, the ColE 1 plasmid in Escherichia coli blocks the action of the RNA primer (activator) by... [Pg.190]

See other pages where Escherichia coli antibiotic resistance is mentioned: [Pg.105]    [Pg.106]    [Pg.396]    [Pg.434]    [Pg.681]    [Pg.11]    [Pg.395]    [Pg.240]    [Pg.315]    [Pg.2]    [Pg.206]    [Pg.222]    [Pg.592]    [Pg.683]    [Pg.145]    [Pg.187]    [Pg.211]    [Pg.24]    [Pg.83]    [Pg.119]    [Pg.113]    [Pg.986]    [Pg.1287]    [Pg.546]    [Pg.75]    [Pg.1]    [Pg.1443]    [Pg.578]    [Pg.113]    [Pg.16]    [Pg.154]    [Pg.250]    [Pg.413]    [Pg.196]   
See also in sourсe #XX -- [ Pg.221 , Pg.222 ]



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Antibiotic resistance

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