Digestion oxygen

Bomb combustion or digestion Oxygen plasma ashing... 

Analytical Procedures. Oxygen difluoride may be determined conveniently by quantitative appHcation of k, nmr, and mass spectroscopy. Purity may also be assessed by vapor pressure measurements. Wet-chemical analyses can be conducted either by digestion with excess NaOH, followed by measurement of the excess base (2) and the fluoride ion (48,49), or by reaction with acidified KI solution, followed by measurement of the Hberated I2 (4). 

Anaerobic ponds are loaded such that anaerobic conditions prevail throughout the Hquid volume. One of the major problems with anaerobic ponds is the generation of odors. The odor problem can frequentiy be eliminated by the addition of sodium nitrate at a dosage equal to 20% of the appHed oxygen demand. An alternative is the use of a stratified facultative lagoon, in which aerators are suspended 3 meters below the Hquid surface in order to maintain aerobic surface conditions, with anaerobic digestion occurring at the lower depths. 

Air Requirement 15 - 20 cfnt per 1,000 cubic feet of digester capacity is adequate. The air supplied must keep the solids in suspension this requirement may exceed the sludge oxidation requirement A dissolved oxygen concentration of 1 to 2 ppm should be maintained in the aerobic digestion tanks. 

The therapeutic utility of systemically administered ASON had been limited by their short plasma half life (sometimes even less than 3 min). This is due to their sensitivity to nuclease digestion. When the first-generation ASON were chemically modified, e.g., by replacing the oxygen in the phosphodiester bond with sulfur (phosphorothiorate) they obtained an increased stability in biological fluids while their antisense effect has been maintained. First-generation agents can be delivered via intravitreal injection, parenterally, by topical cream, enema, and inhaled aerosol. These antisense... 

For several hours after a meal, while the products of digestion are being absorbed, there is an abundant supply of metabolic fuels. Under these conditions, glucose is the major fuel for oxidation in most tissues this is observed as an increase in the respiratory quotient (the ratio of carbon dioxide produced to oxygen consumed) from about 0.8 in the starved state to near 1 (Table 27-1). 

Basso, D., Panozzo, M.P., Fabris, C., Del Favero, G., Meggiato, T., Fogar, P., Meani, A., Faggian, D., Plebani, M., Burlina, A. and Naccarato, R. (1990). Oxygen derived free radicals in patients with chronic pancreatic and other digestive diseases. J. Clin. Pathol. 43, 403-405. 

Dabrowski, A. and Chwiecko, M. (1990). Oxygen radicals mediate depletion of pancreatic sulphydryl compounds in rats with caerulein-induced acute pancreatitis. Digestion 7, 15-19. 

Nonaka, A., Manabe, T., Kyt ku, T., Tamura, K. and Tobe, T. (1990). Changes in lipid peroxide and oxygen radical scavengers in caerulein-induced acute pancreatitis. Imbalance between the offensive and defense systems. Digestion 47, 130-137. 

Parks, D.A., Bulkley, G.B. and Granger, D.N. (1983). Role of oxygen-derived free radicals in digestive tract diseases. Surgery 94, 415-422. 

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