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Breath odor humans

Doty, R.L., Green, P.A., Ram, C. and Yankell, S.L. (1982) Communication of gender from human breath odors relationship to perceived intensity and pleasantness. Horm. Behav. 16, 13-22. Doty, R.L., Kligman, A., Leyden, J. and Omdorff, M.M. (1978) Communication of gender from human axillary odors Relationship to perceived intensity and hedonicity. Behav. Biol. 23, 373-380. [Pg.197]

Odor. Odor is excluded in military appHcations and in the four grades specified for human breathing. The gas is tested by smelling a flowing stream or by smelling a beaker from which a Hquid sample has just evaporated. Oxygen that is produced in modem ASUs employing turbine compressors or nonlubricated piston machines is odorless. [Pg.480]

Normally, only small amounts of acetone are produced in the human body. Diabetics produce larger amounts, sometimes enough to give their breath a sweet odor. [Pg.593]

Toxicology. Tellurium causes garlic odor of the breath and malaise in humans. [Pg.655]

Dangerously toxic by inhalation. Exposure can cause injury to lungs and liver pneumonitis, and strong irritant actions on skin, eyes, and mucous membranes. Other effects are garlic odor of breath, nausea and dizziness. A few minutes exposure to 50-100 ppm concentration of this gas in the air can be lethal to humans. LClo inhalation (rat) 20 mg/m /l hr. [Pg.378]

Human clinical studies Allicin is well absorbed, as indicated by a persistent garlicky odor on the breath, skin, and amniotic fluid of persons after consumption of fresh garlic (83). Because oral comsumption of pure allicin has been shown to significantly increase overall body catabolism of triglycerides, a substantial absorption of allicin is assumed to occur (84). However, the metabolic fate of allicin in the body is not well understood. [Pg.222]

The irritancy properties of isophorone have also been observed in humans exposed occupationally to isophorone. In an industrial hygiene survey, Kominsky (1981) reported that the eye and nose irritation complained of by a screen printer could have been caused by 4-minute exposure to 25.7 ppm isophorone, which was measured in the personal breathing zone while the worker was washing a screen. Lee and Frederick (1981) found that eye, respiratory, and skin irritation were among the complaints of 27/35 workers in a printing plant where isophorone and other solvents (xylene, methylene chloride, and toluene) were used. On the day of measurement, two of the screen printers were found to be exposed to 8-hour TWA concentrations of isophorone of 0.7 and 14 ppm, but it was not clear whether these two individuals were among the workers complaining of irritation. The odor threshold for isophorone in air has been reported to be 0.2 ppm (v/v) (Amoore and Hautala 1983). [Pg.29]

The molecular structures of the ketone bodies acetoacetate, p-hydnoxybutyrate, and acetone are shovvn in Figure 4,64. Acetone is formed by nonerKymatic breakdown of acetoacetate. Between 20 and 75% of the acetone formed can be metabo lized to usable energy by humans. Experiments with rats determined that acetone is metabolized via acetyl-CoA (Kosug cf fl/ 1 56). Unmetaboiiaed acetone is excreted in the urine or released in the breath. The odor of acetone on a person s breath reveals that the person either has uncontrolled diabetes or is fasting. The acetoacetate and 3-liydroxybutyrate formed in the body can be an important source of energy acetone is relatively unimportant. [Pg.236]

Tellurium has no significant biological role. Tellurium and tellurium compounds should be considered to be toxic and need to be handled with care. Organic tellurides have been employed as initiators for living radical polymerization, and electron-rich mono- and di-tellurides possess antioxidant activity. Humans exposed to as little as 0.01 mg/m or less in air develop "tellurium breath", which has a garlic-like odor. This is due to formation of ethyl telluride within the body. [Pg.905]


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See also in sourсe #XX -- [ Pg.191 , Pg.365 ]




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