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Abdominal compartment syndrome

Consider large-volume paracentesis if evidence of abdominal compartment syndrome is secondary to tense ascites... [Pg.114]

A 48-year-old woman who had endoscopic injection of enbucrilate for variceal bleeding rapidly had portal and splenic vein emboli, causing intestinal ischemia secondary to abdominal compartment syndrome, with raised intra-abdominal pressure [30 ]. There were no enbucrilate emboli in the mesenteric arteries. [Pg.1014]

Su WT, Stone DH, Lamparello PJ, Rockman CB (2004) Gluteal compartment syndrome following elective unilateral internal iliac artery embolization before endovascular abdominal aortic aneurysm repair. J Vase Surg 39 672-675... [Pg.13]

Excess adiposity, particularly the abdominal obesity associated with increased waist circumference, is associated with insulin resistance, hypertension, and proinflammatory states. The prevalence of this complex of comorbidities associated with obesity, now referred to as the metabolic syndrome, is reaching epidemic proportions in the United States (Grundy et al., 2004 Roth et al., 2002). Indeed, increased abdominal adiposity is one of a cluster of factors that are used in the diagnosis of metabolic syndrome. Abdominal tissue in the trunk occurs in several compartments, including subcutaneous and intraperitoneal or visceral fat. Visceral fat in particular appears to contribute to perturbed fuel metabolism by at least two mechanisms. First, hormones and free fatty acids released from visceral fat are released into the portal circulation and impact directly on metabolism of the liver. Second, the visceral adipose depot produces a different spectrum of adipocytokines than that produced by subcutaneous fat (Kershaw and Flier, 2004). [Pg.251]


See other pages where Abdominal compartment syndrome is mentioned: [Pg.113]    [Pg.248]    [Pg.248]    [Pg.540]    [Pg.113]    [Pg.248]    [Pg.248]    [Pg.540]    [Pg.202]    [Pg.505]   
See also in sourсe #XX -- [ Pg.248 ]




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