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Transcriptional programming

VR Iyer, MB Risen, DT Ross, G Schuler, T Moore, JCF Lee, JM Trent, LM Staudt, J Hudson Jr, MS Boguski, D Lashkari, D Shalon, D Botstem, PO Brown. The transcriptional program m the response of human fibroblasts to serum. Science 283 83-87, 1999. [Pg.349]

Royzman I, Whittaker AJ, Orr-Weaver TL 1997 Mutations in Drosophila DP and E2F distinguish Gj-S progression from an associated transcriptional program. Genes Dev 11 1999-2011... [Pg.12]

LaTulippe E, Satagopan J, Smith A, Scher H, Scardino P, Reuter V, Gerald WL. Comprehensive gene expression analysis of prostate cancer reveals distinct transcriptional programs associated with metastatic disease. Cancer Res 2002 62 4499-4506. [Pg.410]

Amundson, S. A., M. Bittner, P. Meltzer, J. Trent, and A. J. Fornace Jr. (2001). Physiological function as regulation of large transcriptional programs the cellular response to genotoxic stress. Comp. Biochem. Physiol. B, 129 703-710. [Pg.152]

The G2/M checkpoint acts to ensure that cells that experience DNA damage in G2 or that contain unresolved damage from the previous Gi or S phase do not initiate mitosis. Much like the Gi checkpoint and in contrast to the S checkpoints, cell-cycle arrest or delay that results from the G2 checkpoint involves a combination of acute/transient and delayed/sustained mechanisms. The acute/transient mechanisms involve the rapid posttranslational modification of effector proteins, whereas the delayed/sustained mechanism involves the alteration of transcriptional programs (192). [Pg.161]

Schulze A, Lehmann K, Jefferies HB, McMahon M, Downward J. Analysis of the transcriptional program induced by Raf in epithelial cells. Genes Dev. 2001 15 981-994. [Pg.2219]

Figure 3, Renal stress induces signal transduction and transcriptional programs that are modified by survival factors. The balance between the prosurvival or prodeath aspects of the stress response determines the ultimate fate of the cells. Figure 3, Renal stress induces signal transduction and transcriptional programs that are modified by survival factors. The balance between the prosurvival or prodeath aspects of the stress response determines the ultimate fate of the cells.
Hughes TR, Mao M, Jones AR, Burchard J, Marton MJ, Shannon KW, I-efkowitz SM, Iyer VR, Eisen MB, Ross DT, Schuler G, Moore T, Lee JCE, Trent JM, Staudt LM, Hudson Jr. J, Boguski MS, Lashkari D, Shalon D, Botstein D, Brown PO. 1999. The transcriptional program in the response of human fibroblasts to serum. Science 283 83-87. [Pg.141]


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See also in sourсe #XX -- [ Pg.137 , Pg.342 ]




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