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Renal failure enteral nutrition

Other causes include magnesium-containing antacids in patients with renal insufficiency, enteral or parenteral nutrition in patients with multiorgan system failure, magnesium for treatment of eclampsia, lithium therapy, hypothyroidism, and Addison s disease. [Pg.909]

Nakasaki H, Katayama T, Yokoyama S, Tajima T, Mitomi T, Tsuda M, Suga T, Fujii K. Complication of parenternal nutrition composed of essential amino acids and histidine in adults with renal failure. J Parenter Enteral Nutr 1993 17(l) 86-90. [Pg.683]

Duerksen DR, Papineau N. Electrolyte abnormalities in patients with chronic renal failure receiving parenteral nutrition. J Parenter Enteral Nutr 1998 22(2) 102-4. [Pg.2719]

Claris-Appiani A, ArdissinoGL, DaccoV, et al. Catch-upgrowth in children with chronic renal failure treated with long-term enteral nutrition. JPEN J Parenter Enteral Nutr 1995 19 175-178. [Pg.2589]

TABLE 139—1. Enteral Nutrition Products for Patients with Renal Failure... [Pg.2637]

Phosphorus can be omitted from the nutritional formula of patients receiving PN until the phosphorus level approaches normal (<5 mg/dL). It is prudent to monitor phosphorus concentrations daily and to add phosphorus in small doses once the serum concentration is below 4 mg/dL. Failure to do so can lead to severe hypophosphatemia (see Chap. 49) despite continued renal failure, especially in the patient treated with CRRT. Patients with persistently high serum phosphorus concentrations who have a functional gastrointestinal tract (GIT) can be prescribed phosphate-binding therapy (see Chap. 44) and enteral feedings low in phosphorus to minimize the absorption of exogenous phosphorus. [Pg.2638]


See other pages where Renal failure enteral nutrition is mentioned: [Pg.434]    [Pg.2177]    [Pg.979]   
See also in sourсe #XX -- [ Pg.1513 , Pg.1520 , Pg.1520 ]




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