Radiation carcinogenesis

Cohen, B.L., Surveys of Radon Levels in U.S. Homes as a Test of the Linear-No Treshold Dose-Response Relationship for Radiation Carcinogenesis, this volume (1987). 

A Test of the Linear-No-Threshold Dose-Response Relationship for Radiation Carcinogenesis... 

Baum, J.W. Clonal Theory of Radiation Carcinogenesis, Proceedings of the Symposium on Microdosimetry pp. 12, National Technical Information Service, Springfield, VA, DE 82022044 (1982). 

Brown, J.M., The Shape of the Dose-Response Curve for Radiation Carcinogenesis Extrapolation to Low Doses, Radiation Research 71 34-50 (1977). 

Van Bekkum, D.W. and P. Bentvelzen, The Concept of Gene Transfer-Misrepair Mechanism of Radiation Carcinogenesis May Challange the Risk Estimation For Low Radiation Doses, Health Physics 43 231-237 (1982). 

Continuance of protracted exposure studies to measure carcinogenesis in animal and human cell lines and the role of secondary factors — especially chemical agents — in radiation carcinogenesis (Little 1990)... 

Ananthaswamy, H.N. and W.E. Pierceall. 1990. Molecular mechanisms of ultraviolet radiation carcinogenesis. 

Bowden, G.T., D. Jaffe, and K. Andrews. 1990. Biological and molecular aspects of radiation carcinogenesis in mouse skin. Radiation Res. 121 235-241. 

Coggle, J.E. and J.P. Williams. 1990. Experimental studies of radiation carcinogenesis in the skin a review. Inter. Jour. Radiation Biol. 57 797-808. 

Fry, R.J.M. 1991. Radiation carcinogenesis in the whole-body system. Radiation Res. 126 157-161. 

A dose-response model for radiation carcinogenesis is, however, incomplete in the sense that factors other than dose will govern the response, e.g., dose-rate, host characteristics, and temporal peittems descriptive of time to initiation of effect and of duration of effect. 

Dose-response model. There is incomplete agreement on the most suitable form of dose-response function for low-LET radiation, and there is no present expectation that human observations will prove decisive. Rather, it appears that appropriate models will require a more adequate understanding of radiation carcinogenesis itself. 

Sensitivity of specific target tissues We are confronted with the fact that risk estimates per unit of tissue dose (as currently measured) vary widely among spedfic organs, whether measured in terms of absolute or relative risk, and that these estimates bear no obvious relation to natural incidence. Until we know the basis for these differences, radiation carcinogenesis will not be understood. 

Bair, W.J. (1986). Experimental carcinogenesis in the respiratory tract, page 151 in Radiation Carcinogenesis, Upton, A.C., Albert, R.E., Bums, EJ., and Shore, R.E. Eds. (Elsevier Science Publishing Co. Inc., Elmira, New York). 

Howe, G.R. (1984). "Epidemiology of radiogenic breast cancer," pp. 119 in Radiation Carcinogenesis Epidemiology and Biological Significance, BOICE, J.D., Jr. and Fraumeni, J.F., Jr., Eds. (Raven Press, New York). 

Ullrich, R.L. and Storer, J.B. (1978). Influence of dose, dose-rate and radiation quality on radiation carcinogenesis and life shortening in RFM and BALB/C mice, in Late Biological Effects of Ionizing Radiation, VoL II., Proceedings of a Symposium, Vienna, 1978, (International Atomic Energy Agency, Vienna). 

Upton, A.C. (1967). Comparative observations on radiation carcinogenesis in man and animals. page 631 in Carcinogenesis A Broad Critique. (Williams and Wilkins, Baltimcae). 

Burns, F.J., Uddin, A.N., Wu, F. et al. (2004) Arsenic-induced enhancement of ultraviolet radiation carcinogenesis in mouse skin a dose-response study. Environmental Health Perspectives, 112(5), 599-603. 

Albert RE, Burns FJ, et al., eds. Radiation carcinogenesis. New York, NY Elsevier Science Publishing Co., 335-345. 

Mays CW, Speiss H. 1984. Bone sarcomas in patients given radium-224. In Boice JD, Fraumeni JF, eds. Radiation carcinogenesis epidemiology and biological significance. N.Y. Raven Press, pp 24-1252. 

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