Progressing aging

Wilson MA, Dwyer KD, Roy EJ Direct effects of ovarian hormones on antidepressant binding sites. Brain Res Bull 22 181-185, 1989 Winblad B, Adolfsson R, Carlsson A, et al Biogenic amines in brains with Alzheimer s disease, in Alzheimer s Disease A Report of Progress (Ageing, Vol 19). Edited by Corkin S, Davis KL, Growdon JH, et al. New York, Raven, 1982, pp 25-33 Winker MA Tacrine for Alzheimer s disease. Which patient, what dose JAMA 271 1023-1024, 1994... 

Figure 1.6 Decline in various functional capacities and physiological measurements of human males with progressive age (30-80 years). (Courtesy of Dr. N. W. Shock.)...

Figure 7.36. Diagenetic sequence of textural, mineralogical and geochemical changes for the carbonate sediments of the Mediterranean coast of Israel. In this case the diagenetic sequence is interpreted to be related to the progressive aging of the carbonate sediments. (After Gavish and Friedman, 1969.)...

Though enhancer sensitive neurons exist also outside the mesencephalon, we used the term mesencephalic enhancer regulation to emphasize the key importance of the dopaminergetic neurons, the most rapidly aging neurons of the brain, primarily responsible for the progressive age related decline of behavioral performances. 

Daniel T. Rodgers, Atlantic Crossings Social Politics in a Progressive Age (Cambridge, MA Belknap Press of Harvard University, 1998), 285-286. 

Progressive aging n. In a heat-aging test, stepwise raising of the temperature at preset time intervals. 

Struik showed that many polymers age the same way and that the aging behavior in shear creep is influenced by temperature and loaded stress as well as by the aging time. Based on the general viscoelastic theory for the non-isothermal case of Hopkins [2] and Haugh [3], he proved that this theory and the mathematical formalism can be easily applied to the case of progressive aging at constant temperature. 

J defines now the creep compliance under the influence of progressive aging, which is accessible by experiment X is given by Equation 7. 

Figure 2. Schematic course of creep curves at two fixed values of age and A and during progressive aging.

Let us examine a volume recovery experiment and a creep experiment under progressive aging at the same temperature. Then the shift functions b (A,A ) and aj should be comparable if the prehistories are approximately the same and the change in free volume is the origin of aging effects. 

The creep curves under progressing aging show the general shape as anticipated in Figure 2. At short creep times they have the shape of the equilibrium creep curve... 

Figure 4. Creep curves of PS N 7000 o.W. under the influence of progressive aging at = 90°C after various preconditioning times full line represents the equilibrium creep curve at T. ...

The present contribution has shown that the creep behavior of amorphous polymers under the influence of progressing aging can be well described and predicted under any thermal prehistory applying the multiparameter model based on free volume. The only condition necessary is the knowledge of any measured equilibrium creep curve. For each material the multiparameter model with the given set of parameters allows the prediction of the behavior in volume under any complicated thermal history as well. Introducing some additional postulations, the free volume model is adapted to work at low temperatures, i.e., at temperatures below T. Next, the theory should be extended to measurements at still lower temperatures as well as to some other amorphous polymers. 

After a relatively quick intake of aluminum into the intestinal walls its transition into the blood is significantly slower. In plasma aluminum is bound to approximately 80% to proteins, especially to transferrin. The amount considered currently as normal is 5 p,g/liter. Aluminum can be detected in all human organs. The only organs in which the aluminum concentration rises with progressing age are the lungs and the hilus lymphatic glands. 

FIGURE 16.14 Particle gel showing (from left to right) progressive ageing stages. 

Dementia A progressive neuropsychiatric disorder characterized by deterioration in various cognitive processes such as memory, executive functioning and attention. Can be primary, which is usually found with progressive age or secondary, which is secondary to some other disorder like nutrition deficiency. 

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