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Pregnancy pathophysiology

FIGURE 65-1. Sickle gene inheritance scheme for both parents with sickle cell trait (SCT). A, normal hemoglobin S, sickle hemoglobin. Possibilities with each pregnancy 25% normal (AA) 50% SCT (AS) 25% sickle cell anemia (SS). (From Chan CYJ, Moore R. Sickle cell disease. In DiPiro JT, Talbert RL, Yee GC, et al, (eds.) Pharmacotherapy A Pathophysiologic Approach. 6th ed. New York McGraw-Hill 2005 1856.)... [Pg.1004]

Disseminated gonococcal infection in adults (>45 kg), -/ / A recommended treatment regimen for presumptive C. trachomatis infection during pregnancy, if chlamydial infection has not been ruled out (see Table 121-8 in Pharmacotherapy A Pathophysiologic Approach, seventh edition) Ceftriaxone 1 g IM or IV every 24 hours ... [Pg.510]

Dispersion of physiological concentrations of parathyrine in blood serum influence diurnal rhythm, hourly pulsation, oral load with calcium, exercise, pregnancy, age, and menstrual cycle. There are no sex differences. Medication, resection of a tumor causing hypercalcemia, hypertension, and urolithiasis are the causes of pathophysiological mechanisms that also influence the blood level of parathyrine. Establishing the intact PTH provides sufficient reliability and comparability of the results provided the preanalytic phase is maintained. [Pg.268]


See other pages where Pregnancy pathophysiology is mentioned: [Pg.124]    [Pg.568]    [Pg.98]    [Pg.68]    [Pg.82]    [Pg.15]    [Pg.158]    [Pg.512]    [Pg.78]    [Pg.111]    [Pg.347]   
See also in sourсe #XX -- [ Pg.71 , Pg.469 ]




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