NAD +

Keita B, Nad]o L and K]oller K 1991 Surface characterization of a single crystal of sodium decatungstocerate (IV) by the atomic force microscope Surf. Sc/. Lett. 256 L613... [Pg.1726]

FIGURE 15 5 Structure of NAD the oxidized form of the coenzyme nicotinamide adenine dinucleotide The functional part of the coen zyme is framed in red... [Pg.646]

The reverse reaction also occurs m living systems NADH reduces acetaldehyde to ethanol m the presence of alcohol dehydrogenase In this process NADH serves as a hydride donor and is oxidized to NAD" while acetaldehyde is reduced... [Pg.646]

The NAD -NADH coenzyme system is involved m a large number of biological changes during the enzyme... [Pg.647]

IS the oxidation of lactic acid to pyruvic acid by NAD and the enzyme lactic acid coenzyme NAD ... [Pg.647]

We shall encounter other biological processes m which the NAD" conversion plays a prominent role m biological oxidation-reduction... [Pg.647]

Section 15 11 Oxidation of alcohols to aldehydes and ketones is a common biological reaction Most require a coenzyme such as the oxidized form of nicotin amide adenine dmucleotide (NAD" )... [Pg.655]

All the individual steps are catalyzed by enzymes NAD" (Section 15 11) is required as an oxidizing agent and coenzyme A (Figure 26 16) is the acetyl group acceptor Coen zyme A is a thiol its chain terminates m a sulfhydryl (—SH) group Acetylation of the sulfhydryl group of coenzyme A gives acetyl coenzyme A... [Pg.1070]

Cortisol-Cortisone Conversion. Under normal conditions, this equilibrium slightly favors the oxidized compound. Similarly, the conversion of corticosterone to 11-deoxycorticosterone is also mediated by the liP-hydroxysteroid dehydrogenase enzyme system and requites NAD(P) /NAD(P)H. This conversion is especially important both in the protection of the human fetus from excessive glucocorticoid exposure, and in the protection of distal nephron mineral ocorticoid receptors from glucocorticoid exposure (14). The impairment of this conversion is thought to result in hypertension associated with renal insufficiency (15). [Pg.97]

J. W. Hastie and D. W. BonneU, Molecular Chemist of Inhibited Combustion Systems, Feport NBSIF 80-2169, Nad. Buieau of Standards, Washington, D.C., 1980. [Pg.482]

Standardfor FiquifiedHydrogen Systems at Consumer Sites, Nad. Eke Protect. Assoc, pamphlet no. 50B (ANSI Z292.3), 1973. [Pg.436]

Insects poisoned with rotenone exhibit a steady decline ia oxygen consumption and the iasecticide has been shown to have a specific action ia interfering with the electron transport iavolved ia the oxidation of reduced nicotinamide adenine dinucleotide (NADH) to nicotinamide adenine dinucleotide (NAD) by cytochrome b. Poisoning, therefore, inhibits the mitochondrial oxidation of Krebs-cycle iatermediates which is catalysed by NAD. [Pg.270]

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