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Myocardial infarction subacute

Platelet aggregation appears to be of pivotal importance in several acute or subacute coronary syndromes acute myocardial infarction (MI), unstable angina (UA), postangioplasty ischemia, and poststent-... [Pg.376]

Hayakawa K, Takemura G, Kanoh M, Li Y, Koda M, Kawase Y, et al. Inhibition of granulation tissue cell apoptosis during the subacute stage of myocardial infarction improves cardiac remodeling and dysfunction at the chronic stage. Circulation 2003 108 104-109. [Pg.40]

There were no differences in the activities of serum transaminases, lactate dehydrogenase, or creatine kinase in patients with myocardial infarction who received an 18-hour infusion of urokinase compared with patients who received glucose alone (38), but subacute alterations of liver function tests have been described with streptokinase and anistreplase (39). Unexplained increases in transaminase activities have been reported in almost 25% of patients treated with streptokinase (40). In view of the greater prominence of liver dysfunction with streptokinase than with alteplase it could be wiser to choose alteplase rather than streptokinase in patients with previous impaired hepatic function (41). [Pg.3404]

Figure 2.2 (A) A patient with myocardial infarction of anteroseptal zone in a subacute phase (1) normal recording that displays extension of Q waves up to V6 (qrs). Small changes in the placement of precordial V3-V6 leads have significantly modified the morphology of QRS, now being qR in a lead V6. Therefore, according to the... Figure 2.2 (A) A patient with myocardial infarction of anteroseptal zone in a subacute phase (1) normal recording that displays extension of Q waves up to V6 (qrs). Small changes in the placement of precordial V3-V6 leads have significantly modified the morphology of QRS, now being qR in a lead V6. Therefore, according to the...
Figure 4.66 Above (A) Acute phase of evolving Q-wave myocardial infarction of anteroseptal zone. There is a huge ST-segment elevation, especially in I, VL and from V2 to V5, QRS >0.12 s and morphology of complete RBBB that was not present in previous ECG. (B) Twenty-four hours later RBBB have disappeared and subacute anterior extensive infarction becomes evident. There is ST-segment elevation from V1 to V4. The transient presence of new... Figure 4.66 Above (A) Acute phase of evolving Q-wave myocardial infarction of anteroseptal zone. There is a huge ST-segment elevation, especially in I, VL and from V2 to V5, QRS >0.12 s and morphology of complete RBBB that was not present in previous ECG. (B) Twenty-four hours later RBBB have disappeared and subacute anterior extensive infarction becomes evident. There is ST-segment elevation from V1 to V4. The transient presence of new...
Lopez-Sendon J, Gonzalez A, Lopez de Sa E et al. Diagnosis of subacute ventricular wall rupture after acute myocardial infarction sensitivity and specificity of clinical, hemodynamic and echocardiographic criteria. J Am Coll Cardiol 1992 19 1145. [Pg.317]

Fujimoto, K.L., Tobita, K., Merryman, W.D., Guan, J., Momoi, N., Stolz, D.B., Sacks, M.S., Keller, B.B., Wagner, W.R., 2007b. An elastic, biodegradable cardiac patch induces contractile smooth muscle and improves cardiac remodeling and function in subacute myocardial infarction. Journal of the American College of Cardiology 49, 2292-2300. [Pg.410]


See other pages where Myocardial infarction subacute is mentioned: [Pg.207]    [Pg.207]    [Pg.197]    [Pg.97]    [Pg.420]    [Pg.193]    [Pg.266]    [Pg.47]    [Pg.531]    [Pg.133]    [Pg.38]   
See also in sourсe #XX -- [ Pg.295 ]




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Infarct

Infarct, myocardial

Infarction

Myocardial infarction

Subacute

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