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Isoniazid selective toxicity

Isoniazid, the hydrazide of isonicotinic acid was introduced into medical practice for treating tuberculosis in 1953. Isoniazid exhibits bactericidal action on Mycobacterium tuberculosis. It inhibits the synthesis of mycoUc acid, an important component of the cell membrane of mycobacteria. Mycolic acid is specific only to mycobacteria, and it is the cause of the selective toxicity of the drag with respect to these microorganisms. [Pg.526]

Antidepressants selective serotonin reuptake inhibitors, tricyclic antidepressants Antihypertensives felodipine Antibiotics quinolones, isoniazid Bronchodilators albuterol, theophylline Corticosteroids prednisone Dopa agonists levodopa Herbals ma huang, ginseng, ephedra Nonsteroidal anti-inflammatory drugs ibuprofen Stimulants amphetamines, methylphenidate, caffeine, cocaine Sympathomimetics pseudoephedrine Thyroid hormones levothyroxine Toxicity anticholinergics, antihistamines, digoxin Withdrawal alcohol, sedatives... [Pg.1286]

See other pages where Isoniazid selective toxicity is mentioned: [Pg.527]    [Pg.180]    [Pg.23]    [Pg.1252]    [Pg.1402]    [Pg.219]    [Pg.132]    [Pg.234]    [Pg.1912]    [Pg.123]    [Pg.191]    [Pg.349]   
See also in sourсe #XX -- [ Pg.5 , Pg.266 ]



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