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Glial proliferation

Barbero S, Bajetto A, Bonavia R, Porcile C, Piccioli P, Pirani P, Ravetti JL, Zona G, Spaziante R, Florio T, Schettini G (2002) Expression of the chemokine receptor CXCR4 and its ligand stromal cell-derived factor 1 in human brain tumors and their involvement in glial proliferation in vitro. Ann NY Acad Sci 973 60-69... [Pg.266]

Histopathological findings in fatal cases of lead encephalopathy in children include cerebral edema, altered capillaries, and perivascular glial proliferation. Neuronal damage is variable and may be caused by anoxia (EPA 1986a). [Pg.93]

Fig. 36A-C Glial proliferation in temporal neocortex after ischemia. A Triple-staining for Ibal, NeuN, and BrdU in postischemic day-15 STG. Note Ibal/BrdU double-labeled clusters (arrows) or single cells in the vicinity of NeuN+ neurons (arrowheads). B Double-staining for SI00/3 and BrdU in postischemic day-23 IT. Note an S100/ +/BrdU+ doublet. C A CNP+ oligodendrocyte colabeled for BrdU in the postischemic day-79 IT. Scale bars = 20 pm (A) 10 pm (B) 5 pm (C)... Fig. 36A-C Glial proliferation in temporal neocortex after ischemia. A Triple-staining for Ibal, NeuN, and BrdU in postischemic day-15 STG. Note Ibal/BrdU double-labeled clusters (arrows) or single cells in the vicinity of NeuN+ neurons (arrowheads). B Double-staining for SI00/3 and BrdU in postischemic day-23 IT. Note an S100/ +/BrdU+ doublet. C A CNP+ oligodendrocyte colabeled for BrdU in the postischemic day-79 IT. Scale bars = 20 pm (A) 10 pm (B) 5 pm (C)...
Zhang Z, Guth L (1997) Experimental spinal cord injury Wallerian degenerahon in the dorsal column is followed by revascularizahon, glial proliferation, and nerve regeneration. Exp Neurol 147 159-171. [Pg.678]

Histopathological and morphological findings with fatal encephalopathy presented in the form of cerebral edema, endothelial hypertrophy and hyperplasia, and perivascular glial proliferation. Various ischemic disorders were also reported cell necrosis and neuronal loss in isocortex and basal ganglia... [Pg.442]

Di Giovanni, S., Movsesyan, V, Ahmed, E, et al., 2005. Cell cycle inhibition provides neuroprotection and reduces glial proliferation and scar formation after traumatic brain injury. Proc. Natl. Acad. Sci. USA 102, 8333-8338. Dohi, K., Satoh, K., Mihara, Y, et al., 2006. Alkoxyl radical-scavenging activity of edaravone in patients with traumatic brain injury. J. Neurotrauma 23, 1591-1599. [Pg.706]

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See also in sourсe #XX -- [ Pg.151 ]



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