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Evolution genomic

Zlotnik, A., Yoshie, O., and Nomiyama, H. (2006) The chemokine and chemokine receptor superfamilies and their molecular evolution. Genome Biol. 7, 243. [Pg.45]

Ovcharenko, I., Loots, G. G., Giardine, B. M., et al. (2005) Mulan multiple-sequence local alignment and visualization for studying function and evolution. Genome Res. 15, 184-194. [Pg.253]

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Atsumi, S. et al (2010) Evolution, genomic analysis, and reconstruction of isobutanol tolerance in Escherichia coli. Mol Syst. Biol, 6, 449. [Pg.175]

Luchetti A, Mantovani B. Conserved domains and SINE diversity during animal evolution. Genomics 2013 102(4) 296-300. [Pg.339]

Montes G, Diaz Mejia JJ, Perez Rueda E, Segovia L.The hidden universal distribution of amino acid biosynthetic networks a genomic perspective on their origins and evolution. Genome Biol 2008 9 R95. [Pg.467]

Ramagopalan, S. V., Heger, A., Berlanga, A. J., et al. 2010. A ChIP-seq defined genome-wide map of vitamin D receptor binding Associations with disease and evolution. Genome Res, 20 1352-60. [Pg.124]

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In this section, two models of development were presented, a complex model consisting of a multioperon genome and a cytoplasm, and a simple model based on random Boolean networks. The simpler model was explained in more detail, as it is the basis for the extended example described here. This model utilizes both development and evolution to get to a cell that can develop into a multicellular organism able to seek a chemical trace. [Pg.322]

A steady state GA is used for evolution. Evolution is applied to the genome of the organism. An example is shown in Table 10.1. The purpose of evolution... [Pg.322]


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See also in sourсe #XX -- [ Pg.91 ]




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Genome evolution

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