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Chloramphenicol bacteriostatic effects

Chloramphenicol is a bacteriostatic agent that binds to the 508 ribosomal subunit and inhibits the transpeptidation in protein synthesis. While this agent is not widely used to treat staphylococcal infection, resistance to chloramphenicol is due to inactivation of the antibiotic by chloramphenicol acetyltransferase enzyme (CA7). Macrolides, such as erythromycin and oleandomycin lincosamides, such as lincomycin and clindamycin and streptogramin antibiotics also have a bacteriostatic effect on Staphylococcus spp. by binding to their 508 ribosomal subunit, arresting protein synthesis, but resistance to these antibiotics is also prevalent. Rifampin has also been used to treat staphylococcal infections, but when used alone, resistant strains quickly arise. [Pg.185]

See other pages where Chloramphenicol bacteriostatic effects is mentioned: [Pg.112]    [Pg.311]    [Pg.107]    [Pg.10]    [Pg.229]    [Pg.88]    [Pg.1933]    [Pg.311]    [Pg.349]    [Pg.349]    [Pg.20]    [Pg.447]    [Pg.1503]    [Pg.243]    [Pg.29]    [Pg.709]    [Pg.711]    [Pg.30]    [Pg.43]    [Pg.489]    [Pg.141]    [Pg.94]    [Pg.94]    [Pg.29]   
See also in sourсe #XX -- [ Pg.311 ]

See also in sourсe #XX -- [ Pg.311 ]

See also in sourсe #XX -- [ Pg.29 , Pg.311 ]



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