Hydrophobicity cell surface

Rosenberg, M., and Doyle, R. J. (1990). Microbial cell surface hydrophobicity. History, measurement and significance. In "Microbial Cell Surface Hydrophobicity" (R. J. Doyle and M. Rosenberg, eds), pp. 1-37. ASM Press, Washington, D.C. 

Huysman, F. Verstraete, W. (1993a). Water-facilitated transport of bacteria in unsaturated soil columns influence of cell surface hydrophobicity and soil properties. 

Lemke, M. J., P. F. Churchill, and R. G. Wetzel. 1995. Effect of substrate and cell surface hydrophobicity on phosphate utilization in bacteria. Applied Environmental Microbiology 61 913-919. 

Busscher HJ, Sjollema J, Van der Mei HC (1990) Relative importance of surface free energy as a measure of hydrophobicity in bacterial adhesion to solid surfaces. In Doyle RJ, Rosenberg M (eds) Microbial Cell Surface Hydrophobicity. American Society for Microbiology,... 

Alexander, H., Bertrand, R, Charpentier, C. (1998). Ethanol induced yeast film formation with cell surface hydrophobicity as a major determinant. Food Technot. Biotechnol., 36, 27-30. 

Marshall, K.C. and Cruickshank, R.H., 1973. Cell surface hydrophobicity and the orientation of certain bacteria at interfaces. Arch. Mikrobiol., 91 29--40. 

Adherence to host tissue is achieved by a combination of specific and non-specific mechanisms. Specific adherence mechanisms include ligand-receptor interactions, while non-specific mechanisms include electrostatic forces, aggregation and cell surface hydrophobicity. Non-specific interactions are the primary interaction (occurring over longer distances) involved in the adherence process and are reversible. However, non-specific adherence is consequently deemed irreversible as a result of specific mechanisms that involve the ability of the yeast to recognize a variety of host cell receptors/ ligands using cell surface molecules. 

Unlike Gram-positive bacteria, Gram-negative bacteria such as Pseudomonas have an outer membrane. The outer membrane has been shown to play a role in the protection of the cell from solvent toxicity. Ions such as Mg or Ca stabilize the organization of the outer membrane and contribute to solvent tolerance. Low cell surface hydrophobicity caused by changes in the lipopolysaccharide (LPS) content has been reported to serve as a defensive mechanism. " It has also been reported that the porins which are embedded in the outer membrane are relevant to solvent tolerance. ... 

The adhesion of cells to surfaces is different from that of proteins and DNA. Cell adhesion and spreading are believed to depend primarily on the hydrophobicity of both cells and surfaces. Cell surface hydrophobicity is usually associated with the presence of fibrillar structures oti cell surfaces and specific cell wall proteins. Adhesion, spreading, and growth of mammalian cells are generally promoted on hydrophilic surfaces ( static 30 40°) [10], while those of bacterial... 

Zhao Z, Selvam A,Wong JW. Effects of rhamnolipids on cell surface hydrophobicity of PAH degrading bacteria and the biodegradation of phenanthrene. BioresourTechnol 2011 102 3999-4007. 

Di Ciccio P, et al. Biofilm formation by Staphylococcus aureus on food contact surfaces relationship with temperature and cell surface hydrophobicity. Food Control 2015 50 930-6. 

When A. brasilense was cultured in Luria-Bertani rich medium, the surface composition varied during growth, as illustrated by Fig. 40. Modeling the composition in terms of molecular compounds indicated that the protein concentration increased, from 30 (exponential phase cells) to 50% (stationary phase cells), concomitantly with a decrease in the polysaccharide concentration, from 60 to 35%. These modifications were related to a change in cell-surface hydrophobicity, the water contact angle measured on cell lawns increasing from 20 to 60° (Fig. 40). No difference of electrophoretic mobility was detected between cells harvested in the exponential... 

CMC, they caused changes in the bacterial outer membrane protein composition yet did not affect the LPS component. Chang et al. (2009) demonstrated that the cell-surface hydrophobicity was enhanced by the acciunulation at the cell surface of different fatty acids during growth on hydrocarbon in R. erythropolis NTU-1. A significant correlation between the modification of the cell surface by saponins and the degree of hydrocarbon biodegradation was reported by Kaczorek et al. (2008). 

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