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Cancer risk assessment paradigm

Use of this proposed approach represents a significant paradigm shift in noncancer risk assessment and is inconsistent with basic biological principles. Earlier in this chapter we discussed the fact that it is unlikely for many carcinogens to operate with a linear dose-response. Although it is possible for a single molecule to interact with DNA to increase cancer risk, carcinogenesis is not a multistep process, and protective controls are in place to limit tumor formation. [Pg.675]

Borm, P.J.A., Schins, R.P.F. and Albrecht, C. (2004) Mini review inhaled particles and lung cancer. Part B paradigms and risk assessment. International Journal of Cancer, 110, 3-14. [Pg.241]

See other pages where Cancer risk assessment paradigm is mentioned: [Pg.184]    [Pg.176]    [Pg.586]    [Pg.587]    [Pg.588]    [Pg.659]    [Pg.140]    [Pg.157]    [Pg.501]    [Pg.401]    [Pg.18]    [Pg.2220]    [Pg.176]    [Pg.189]    [Pg.392]   
See also in sourсe #XX -- [ Pg.18 ]



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