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Cancer rate, colon, correlation

Figure 7- Correlation between colon cancer rate and the intake of cereals in 37 countries. (Adapted from Ref. 37.)... Figure 7- Correlation between colon cancer rate and the intake of cereals in 37 countries. (Adapted from Ref. 37.)...
Example 1 (continued). In the ecological study conducted by Kono et al., Pearson correlation coefficients were calculated for annual data on consumption of selected nutrients and food and colon cancer rate with a lag time of 20 years. A positive correlation was found between consumption of fat (r = 0.97 for men and 0.98 for women), meat (r = 0.98 for men and 0.97 for women) and alcohol (r = 0.96 for men and 0.98 for women) and cancer incidence similar results were found for cancer mortality. Negative correlations were found for consumption of miso and cereal. The high correlation found for consumption of alcohol and fat may be somewhat misleading because there were only small variations in consumption whereas there was a much larger variation in average intake of meat and cancer incidence. [Pg.613]

Studies of the incidence of and mortality from colon cancer at the international level suggest an association of this neoplasm with total dietary fat (33,34). Lui et al (35), studying the disappearance rate of food and mortality from colon cancer between the years 1967-1973 in 20 industrialized countries, concluded that there was a direct correlation of this tumor type and the per capita intake of total fat, saturated and monounsaturated fat and cholesterol. Furthermore, fiber intake was inversely correlated with colon cancer in these studies. [Pg.172]

International comparitive studies show a strong correlation between per capitut disappearance of fat and rates of colon cancer (Willett, 2001a Kushi and Giovannucci, 2002). Early animal studies suggested that dietary fat plays an important role in the initiation and promotion of colon tumorigenesis. However, later evidence showed that total energy intake, rather than fat intake, was more likely to influence tumor development (Howe et al., 1997). [Pg.605]

The expression of Pgp, as detected by immunohistochemistry (IHC), has been shown to display a positive correlation with increased relapse rate in osteosarcoma (14). This prognostic significance of Pgp was unrelated to other features of the tumor such as chemotherapy-induced necrosis, which is currently the most important predictor of disease-free survival. It is of interest that in this study the relationship between Pgp and tumor relapse after chemotherapy could not be linked to increased drug efflux from the tumor cells. The chemotherapy used was composed of drugs that are not normally considered to be substrates for Pgp. Therefore, at least in osteosarcoma and perhaps also in colon and breast cancer, the presence of Pgp may not simply be a marker of tumor chemosensi-tivity, but also a sign of tumor aggressiveness (15). [Pg.5]


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Colon cancer

Colonic cancer

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