Anticlinicity

Anticlinal traps which are the result of ductile crustal deformations... 

In many oil and gas fields throughout the world hydrocarbons are found in fault bound anticlinal structures. This type of trapping mechanism is called a combination trap. 

A secondary feature is the development of rollover anticlines which form as a result of the downward movement close to the fault plane which decreases with increasing distance from the plane. Rollover anticlines may trap considerable amounts of hydrocarbons. 

Figure 5.7 Geometry of growth faulting and resulting anticline (rollover) (after Petroleum Flandbook, 1983)...

Folds are features related to compressional, ductile deformation (Fig. 5.10). They form some of the largest reservoir structures known. A fold pair consists of anticline and syncline. 

Calculate the rotational barrier between the anti and anticlinal forms of N-butane using the AMI (or PM3 if you prefer) and HF/6-31G(d) model chemistries. Use the results for the anti form that you obtained in Exercise 6.1. Note that the anticlinal form is a transition structure you will find the Opt TS,CalcFC] keyword helpful in optimizing this structure. 

Sail-related (raps—formed when the plastic salt formations deform into domelike structures under the overburden forces of the beds above the salt beds. Such plastic flowing (and bulging) of the salt beds deforms the rock formations above producing anticline structures and faults in the rock formation astride the domelike structures (see Figures 2-49 and 2-50). 

In contrast, the fluoride ion induced cyclization proceeds via an anticlinal approach which minimizes steric congestion between the allylsilane and the substituent in the 3-position of the cyclohexenone50. Thus, the product having a cis relationship between the vinyl group and R2 at the ring junction is formed preferentially 48 " 51. The controlling element is the substituent R2. In its absence (R2 — H) the diastereoselectivity disappears (see Table 1). 

All the modifications of the a form would present, perpendicular to the b axis, macromolecular bilayers including all isoclined chains. A regular succession of bilayers with anticlined helices would correspond to the limiting ordered modification (a2), while a statistical succession of bilayers would correspond to the limiting disordered modification (a ) [40]. 

In the a modifications of i-PP, bilayers of macromolecules are stacked one on the top of the other in such a way that the top layer on one bilayer and the bottom layer of the bilayer in contact are made up of helices which are enantiomorphous. such helices are regularly anticlined for the ordered a2 modification, more or less at chance isoclined or anticlined for the disordered modifications. 

In the pyranoses in the chair conformations, the vicinal hydroxyl groups can exist only in the anticlinal or gauche orientations. The gauche arrangement of a-glycols is normally encountered as diequatorial or axial-... 

The EMSIL obtained with the purified EPG on transverse sections of barley leaf epidermal cells taken pependicular to the long axis of the cells and anticlinal to the leaf surface, revealed that EPG substrate is localized primarily in the cell comers and middle lamella of these cells (Fig. 1). 

Figure 1. Transverse section of barley leaf epidermal cells taken perpendicular to the long axis of the cells and anticlinal to the leaf surface. The section has been labeled by the EMSIL technique (see Methods) utilizing purified C. sativus endopolygalacturonase and monoclonal antibody EPG-4, which is specific for this enzyme, in order to localize the substrate of the enzyme at the typical site penetrated by the fungal pathogen. Bar = 1 pm. Inset Comparable cell wall region as in Fig. 1 but labeled with monoclonal antibody JIM 5 to localize non-esterified pectin. Bar = 1 pm. Note the identical labeling patterns obtained with either method.

Figure 2. Advanced stage of barley leaf penetration by C. sativus. The pathogen has penetrated the anticlinal cell wall junction between two host epidermal cells (e). The fungal appressorium (a) is visible above the cell comer. The host cell comer matrix has been displaced by an enlarged hyphal element (h) situated between the thin cell walls of the host epidermal cells. The host epidermal cell walls have been densely labeled with the cellulase-gold probe. An intercellullar hyphal element (ih) is present within the penetrated host cell. Bar = 1 pM.

FIG. 1. Shoot apical meristem structure. The shoot apical meristem consists of three clonally distinct cell layers (LI, L2, L3). LI and L2 represent tunica layers, L3 represents the corpus. Cell divisions in LI and L2 are exclusively anticlinal, cell divisions in the L3 occur in all planes. The central zone harbours the stem cells and is surrounded by the peripheral zone where organ primordia are initiated. 

The chiral sites which are able to rationalize the isospecific polymerization of 1-alkenes are also able, in the framework of the mechanism of the chiral orientation of the growing polymer chain, to account for the stereoselective behavior observed for chiral alkenes in the presence of isospecific heterogeneous catalysts.104 In particular, the model proved able to explain the experimental results relative to the first insertion of a chiral alkene into an initial Ti-methyl bond,105 that is, the absence of discrimination between si and re monomer enantiofaces and the presence of diastereoselectivity [preference for S(R) enantiomer upon si (re) insertion]. Upon si (re) coordination of the two enantiomers of 3-methyl-l-pentene to the octahedral model site, it was calculated that low-energy minima only occur when the conformation relative to the single C-C bond adjacent to the double bond, referred to the hydrogen atom bonded to the tertiary carbon atom, is nearly anticlinal minus, A- (anticlinal plus, A+). Thus one can postulate the reactivity only of the A- conformations upon si coordination and of the A+ conformations upon re coordination (Figure 1.16). In other words, upon si coordination, only the synperiplanar methyl conformation would be accessible to the S enantiomer and only the (less populated) synperiplanar ethyl conformation to the R enantiomer this would favor the si attack of the S enantiomer with respect to the same attack of the R enantiomer, independent of the chirality of the catalytic site. This result is in agreement with a previous hypothesis of Zambelli and co-workers based only on the experimental reactivity ratios of the different faces of C-3-branched 1-alkenes.105... 

While the existence of the anticlinic minimum in conventional SmC materials has been suggested by experiments with DOBAMBC,17 by far the majority of tilted smectics only exhibit the synclinic structure of the SmC or SmC under normal conditions. When the mesogenic compound is unichiral (i.e., enantiomerically pure) or enantiomerically enriched, a macroscopic polarization with the same orientation in each layer is produced, as discussed above. In the SmCA phase of MHPOBC, however, the anticlinic structure is the global minimum structure, as shown at the bottom of Figure 8.9. In this case, given that the chirality of the molecules is fixed, the polarization must alternate... 

Figure 8.9 Synclinic and anticlinic layer interface configurations, differing in relative azimuthal angle <(> between director orientations for pair of adjacent layers, are illustrated. It appears that many, if not all, SmC and SmC materials possess wells for both synclinic and anticlinic configurations. Materials for which antichnic well is global minimum for system in some temperature...

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