Vascular homeostasis

Vascular access ports Vascular homeostasis Vasectomy Vasicine [6159-55-3]... 

Lefebvre P, Chinetti G, Fruchart J-C et al (2006) Sorting out the roles of PPARa in energy metabolism and vascular homeostasis. J Clin Invest 116 571-580... 

Evidence also suggests that stem cells play key roles in the adult heart s ability to dynamically repair itself and its vessels and in the body s ability to maintain vascular homeostasis. 

As mentioned above, EPCs appear to be important in vascular homeostasis [44]. Accordingly, Werner... 

Bazzoni G, Dejana E (2004) Endothelial cell-to-cell junctions molecular organization and role in vascular homeostasis. Physiol Rev 84 869-901... 

S. A., Sawyer, P. N., Biophysical Mechanisms in Vascular Homeostasis and Intravascular Thrombosis, P. N. Sawyer, Ed., pp. 306-313, Apple-ton-Century-Crofts, New York, 1965. 

Radomsid MV, Moncada S. Regulation of vascular homeostasis by nitric oxie. Uiromb Haemost 70 36-41,1993. 

History of the discovery ofnitric oxide as an endogenous regulator of vascular homeostasis... 

Other milestones in research on the biological significance ofNO in vascular homeostasis were the description of the molecular pathway of NO biosynthesis by NO synthase fixrm a semi-essential amino acid, L-arginine (Palmer et al, 1988), and the identification of this enzyme and its role as modulator of platelet function in human platelets (Radomski et al, 1990a). 

Fleming I. Cytochrome P-450 enzymes in vascular homeostasis. Circulation Res 2001 89 753-762. 

NO is also able to modulate vascular functions by altering the expression of genes encoding certain endothelial proteins as well as the genes that encode them. These effects underscore the significance of endothelium-derived NO, both in short-term and long-term vascular homeostasis. 

Symposium Role of Prostaglandins in Vascular Homeostasis, Fed. Proc. 35 (1976) 2358-2392. 

Endothelial nitric oxide synthase (eNOS). The enzyme responsible for nitric oxide production in human endothelium. Nitric oxide (NO) is a gas that relaxes vascular smooth muscle cells, induces vasodilatation and is critical for global vascular homeostasis. cNOS is a homodimer enzyme that couples oxygen consumption to L-arginine use to produce NO. In its uncoupled form, cNOS is rendered into a source of superoxides instead of NO. 

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