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UGT polymorphisms

Riedmaier S, Klein K, Hofmann U, Keskitalo JE, Neuvonen PJ, Schwab M, Niemi M, Zanger UM (2010) UDP-glucuronosyl-transferase (UGT) polymorphisms affect atorvastatin lactonization in vitro and in vivo. Clin Pharmacol Ther 87 65-73... [Pg.87]

Krishnaswamy S, Hao Q, Al-Rohaimi A, et al. UDP glucuronosyltransferase (UGT) 1A6 pharmacogenetics II. Functional impact of the three most common non-synonymous UGT1A6 polymorphisms (S7A, T181A, and R184S). J Pharmacol Exp Ther 2005 313(3) 1340-1346. [Pg.117]

FujitaK, Ando Y, NagashimaF, etal. Genetic linkage of UGTl A7 and UGT 1A9 polymorphisms to UGT1A1 6 is associated with reduced activity for SN-38 in Japanese patients with cancer. Cancer Chemother Pharmacol 2007 60(4) 515-22. [Pg.82]

Huang CK, Dulau A, Su-RickCJ, etal. Validation ofrapid polymerase chain reaction-based detection of all length polymorphisms in the UGT lAl gene promoter. Diagn Mol Pathol 2007 16(l) 50 3. [Pg.82]

UGTIAI is the UGT principally responsible for bilirubin glucuronidation. There are 60 rare mutations in the UGTIAI gene known to date, but only a few of them occur with a sufficient frequency (> 1%) to represent polymorphisms. [Pg.165]

See other pages where UGT polymorphisms is mentioned: [Pg.88]    [Pg.282]    [Pg.734]    [Pg.510]    [Pg.88]    [Pg.282]    [Pg.734]    [Pg.510]    [Pg.1266]    [Pg.43]    [Pg.321]    [Pg.126]    [Pg.236]    [Pg.236]    [Pg.243]    [Pg.247]    [Pg.251]    [Pg.63]    [Pg.64]    [Pg.281]    [Pg.63]    [Pg.220]    [Pg.69]    [Pg.116]    [Pg.498]    [Pg.241]    [Pg.250]    [Pg.1266]    [Pg.472]    [Pg.508]    [Pg.88]    [Pg.626]    [Pg.25]    [Pg.74]    [Pg.74]    [Pg.80]    [Pg.26]    [Pg.28]    [Pg.349]    [Pg.164]    [Pg.170]    [Pg.59]    [Pg.886]    [Pg.210]    [Pg.212]    [Pg.1469]    [Pg.1488]   
See also in sourсe #XX -- [ Pg.241 ]



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UGTs

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