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Synapse development

Sternfeld, M., Ming, G., Song, H., Sela, K., Poo, M. and Soreq, H. (1998). Acetylcholinesterase enhances neutrite growth and synapse development through alternative contributions of its hydrolytic capacity, core protein, and variable C termini. Journal of Neuroscience 18 1240-1249. [Pg.166]

Fat facets Deubiquitinating enzyme controlling synapse development in Drosophila... [Pg.736]

Lu B, Figurov A. Role of neurotrophins in synapse development and plasticity. Rev Neurosci 1997 8 1-12. [Pg.150]

Introduction on the Use of the Drosophila Embry-onic/Larval Neuromuscular Junction as a Model System to Study Synapse Development and Function, and a Brief Summary of Pathfinding and Target Recognition... [Pg.456]

Plasticity and Second Messengers During Synapse Development... [Pg.456]

Liebl FLW, Chen K, Karr J, Sheng Q, Featherstone DE. 2005. Increased synaptic microtubules and altered synapse development in Drosophila sec8 mutants. BMC Biology 3 27. [Pg.230]

Slone J, Daniels J, Amrein H (2007) Sugar receptors in Drosophila. Curr Biol 17 1809-1816 Sone M, Suzuki E, Hoshino M, Hou D, Kuromi H, Fukata M, Kuroda S, Kaibuchi K, Nabeshima Y, Hama C (2000) Synaptic development is controlled in the periactive zones of Drosophila synapses. Development 127 4157-4168... [Pg.196]

Suzuki S, Kiyosue K, Hazama S, et al. Brain-derived neurotrophic factor regulates cholesterol metabolism for synapse development. Neurosci. 2007 27 6417-6427. [Pg.241]

Littleton J.T., Bellen H.J., and Perin M.S. 1993. Expression of synaptotagmin in Drosophila reveals transport and localization of synaptic vesicles to the synapse. Development WSi 1077-1088. [Pg.198]

Two specialties of the nervous system are speed and localization, accompHshed using highly developed electrical signaling and close cellular apposition. At specialized points of communication, such as the synapse and the neuromuscular junction, the cells are separated by a nanometer or less. [Pg.515]

Selected for clinical trials as a compound to calm agitated patients, imipramine was relatively ineffective. However, it was observed to be effective in the treatment of certain depressed patients (38). Early studies on the mechanism of action showed that imipramine potentiates the effects of the catecholamines, primarily norepinephrine. This finding, along with other evidence, led to the hypothesis that the compound exerts its antidepressant effects by elevating norepinephrine levels at central adrenergic synapses. Subsequent studies have shown that the compound is a potent inhibitor of norepinephrine reuptake and, to a lesser extent, the uptake of serotonin, thus fitting the hypothesis that had been developed to explain the antidepressant actions ofMAOIs. [Pg.467]

ChEs control the duration of ACh-mediated action on post-synaptic receptors in cholinergic synapses, and have non-hydrolytic roles in nervous systems development and plasticity. [Pg.357]

Long-term potentiation and depression of glutamatergic synapses are involved in many models for brain function and development. A key factor in the plasticity is a change in the AMPA and kainate... [Pg.126]

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See also in sourсe #XX -- [ Pg.165 ]



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