Pathologic Physiology of EHL

Ideally, diseases are classified on the basis of etiologic factors. It is the consensus of students in the field of lipid metabolism that such a goal has not been reached with regard to the various disorders of lipid metabolism and the term essential hyperlipemia reflects this ignorance. However, significant advances from a purely [Pg.452]

P ui/ltlllllUllllll iLjimimiiii P tlMMIHtllll HimMiiiiiiii (illlllllltlllB IIIIIIIIIIIINIi [Pg.452]

Familial fat-induced hyperlipemia. Plasma creamy Xanthomatosis eruptive, tuberous Hepato-splenomegaly Chylomicrons on any fat intake Post-heparin lipolytic activity low Glucose tolerance normal. [Pg.452]

Familial Hyper-/8-Lipoproteinemia (Familial Hypercholesterolemia). Plasma clear Xanthomatosis Tendon, tuberous, xanthelasmas Arcus Expression early .Severity increases with time Postheparin lipolytic activity normal Glucose tolerance normal Atheromatosis. [Pg.452]

Familial Hyper-/8- and Pre-/8-Lipoproteinemia ( Familial Hypercholesterolemia with Hyper-glyceridemia ). Plasma clear, cloudy or milky Xanthomatosis Tuberous, tendon, plane Arcus Post-heparin lipolytic activity normal Glucose tolerance abnormal Carbohydrate inducible Atheromatosis. [Pg.452]

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