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Organophosphorus-induced delayed polyneuropathy OPIDP

Johnson, M.K., Glinn, P. (1990). Neuropathy target esterase (NTE) and organophosphorus-induced delayed polyneuropathy (OPIDP) recent advances. Toxicol. Lett. 82/83 459-63. [Pg.885]

The term delayed neurotoxicity may be used to describe any type of toxicity to the nervous system involving a delay between the precipitating chemical exposure and the appearance of neurological signs or symptoms. However, this designation usually refers to organophosphorus (OP) compound-induced delayed neurotoxicity (or delayed neuropathy) (OPIDN), also known as OP compound-induced delayed polyneuropathy (OPIDP). [Pg.1886]

Organophosphorus compounds (OPC) cause two major toxic effects. The first is the well-known acute toxicity initiated by inhibition of acetylcholinesterase (AChE) with subsequent accumulation of acetylcholine at nerve endings. The second effect is organophosphate-induced delayed polyneuropathy (OPIDP) (ataxia and paralysis appearing 2-3 weeks after exposure) associated with inhibi-... [Pg.247]

See other pages where Organophosphorus-induced delayed polyneuropathy OPIDP is mentioned: [Pg.716]    [Pg.716]    [Pg.1219]    [Pg.1372]    [Pg.573]   
See also in sourсe #XX -- [ Pg.2 , Pg.53 ]



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