Myocardial infarction remodeling after

Britten MB, Abolmaali ND, Assmus B, et al. Infarct remodeling after intracoronary progenitor cell treatment in patients with acute myocardial infarction (TOPCARE-AMI) mechanistic insights from serial contrast-enhanced magnetic resonance imaging. Circulation 2003 108 2212-221 8,... [Pg.437]

St. John Sutton MG, Sharpe N. Left ventricular remodeling after myocardial infarction. Pathophysiology and therapy. Circulation 2000 101 2981-2988. [Pg.369]

Jain M, DerSimonian H, Brenner DA, et al. Cell therapy attenuates deleterious ventricular remodeling and improves cardiac performance after myocardial infarction. Circulation 2001 ... [Pg.450]

Harada M, Qin Y Takano H, et al. G-CSF prevents cardiac remodeling after myocardial infarction by activating the Jak-Stat pathway in cardiomyocytes. Nat Med 2005 I 1(3)305-3 I I. [Pg.450]

Matsushima, S., Ide, T., Yamato, M., Matsusaka, H., Hattori, F., Ikeuchi, M., Kubota, T., Sunagawa, K., Hasegawa, Y., Kurihara, T., Oikawa, S., Kinugawa, S., and Tsutsui, H. 2006. Overexpression of mitochondrial peroxiredoxin-3 prevents left ventricular remodeling and failure after myocardial infarction in mice. Circulation 113 1779-1786. [Pg.152]

Chareonthaitawee, P., Christian, T.F., Hirose, K., Gibbons, R.J., and Rumberger, J.A. 1995. Relation of initial infarct size to extent of left ventricular remodeling in the year after acute myocardial infarction. J. Am. Coll. Cardiol. 25 567-573. [Pg.260]

Ju, H., Zhao, S., Davinder, S.J., and Dixon, I.M. 1997. Effect of ATI receptor blockade on cardiac collagen remodeling after myocardial infarction. Cardiovasc. Res. 35 223-232. [Pg.262]

Pfeffer, J.M., Fischer, T.A., and Pfeffer, M.A. 1995. Angiotensin-converting enzyme inhibition and ventricular remodeling after myocardial infarction. Amur Rev. Physiol. 57 805-826. [Pg.264]

Takemura G, Fujiwara H. Role of apoptosis in remodeling after myocardial infarction. Pharmacol Ther 2004 104 1-16. [Pg.33]

Sam F, Sawyer DB, Chang DL, Eberli FR, Ngoy S, Jain M, et al. Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart. Am J Physiol Heart Circ Physiol 2000 279 H422-H428. [Pg.40]

Palojoki E, Saraste A, Eriksson A, Pulkki K, Kallajoki M, Voipio-Pulkki LM, et al. Cardiomyocyte apoptosis and ventricular remodeling after myocardial infarction in rats. Am J Physiol Heart Circ Physiol 2001 280 H2726-H2731. [Pg.40]

Pfeffer MA, Braunwald E. Ventricular remodeling after myocardial infarction. Experimental observations and clinical implications. Circulation 1990 81 1161-1172. [Pg.40]

Anversa P, Olivetti G, Capasso JM. Cellular basis of ventricular remodeling after myocardial infarction. Am J Cardiol 1991 68 7D-16D. [Pg.40]

Palmen M, Daemen MJ, Bronsaer R, Dassen WR, Zandbergen HR, Kockx M, et al. Cardiac remodeling after myocardial infarction is impaired in IGF-1 deficient mice. Cardiovasc Res 2001 50 516-524. [Pg.40]

Yarbrough WM, Mukherjee R, Escobar GP, Sample JA, McLean JE, Dowdy KB, et al. Pharmacologic inhibition of intracellular caspases after myocardial infarction attenuates left ventricular remodeling A potentially novel pathway. J Thorac Cardiovasc Surg 2003 126 1892-1899. [Pg.40]

Saitoh T, Nakajima T, Takahashi T, Kawahara K. Changes in cardiovascular function on treatment of inhibitors of apoptotic signal transduction pathway in left ventricular remodeling after myocardial infarction. Cardiovasc Pathol 2006 15 130-138. [Pg.42]

Antagonistic effects of cyclo-oxygenase inhibitors (indo-metacin or aspirin) have been repeatedly reported both in hypertension and in heart failure, strongly suggesting that there may be prostaglandin participation in the clinical response to ACE inhibitors (103,104). In animals, although not in all experimental models, aspirin can attenuate the beneficial effects of ACE inhibitors on ventricular remodelling after myocardial infarction. [Pg.232]

McKay RG, Pfeffer MA, Pasternak RC, Markis JE, Come PC, Nakao S, Alderman JD, Ferguson JJ, Safian RD, Grossman W (1986) Left ventricular remodeling after myocardial infarction a corollary to infarct expansion. Circulation 74 693-702... [Pg.374]

V. Marangelli, C. Memmola, M.S. Brigiani, L. Boni, M.G. Biasco, D. Scrutinio, S. Iliceto and P. Riz-zon, Early administration of verapamil after thrombolysis in acute anterior myocardial infarction. Effect on left ventricular remodeling and clinical outcome. VAMI Study Group. Verapamil Acute Myocardial Infarction, Ital Heart J. 1(5), 336-343 (2000). [Pg.75]

A beneficial effect of thyroid hormone on left ventricular remodeling is also reported. In an experimental model of acute myocardial infarction in rats, a decline in serum T3 was observed associated with left ventricular dysfunction and changes in T3 responsive genes. Treatment with high doses of T3 for three weeks resulted in improved cardiac function with normalization of most of the changes in gene expression.89 Similarly, administration of DITPA (a thyroid analogue) after myocardial infarction in rabbits was associated with improved cardiac function.90... [Pg.87]

K. W. Mahaffey, T.E. Raya, G.D. Pennock, E. Morkin and S. Goldman, Left ventricular performance and remodeling in rabbits after myocardial infarction effects of a thyroid hormone analogue, Circulation 91(3), 794-801 (1995). [Pg.97]

M.A. Pfeffer, Left ventricular remodeling after acute myocardial infarction, Anna Rev Med 46,455-466 (1995). [Pg.163]

After the value of drugs has been praised, a very simple remedy should not be forgotten Exercise rehabilitation. This modality has been found to reduce mortality after a myocardial infarction278 and in heart failure.279 A host of beneficial effects has been cited of which preconditioning may not be the least. Very recently, prior training has been shown to improve outcome of myocardial infarction in the rat, probably through improvement in remodeling.280... [Pg.184]

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