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In fetus

Nakamura T, Noda T, Saitoh M, Morita S (1993) Determination of di- and mono-n-butyltin compounds in fetuses and some organs from pregnant rats treated Mth di-rt-butyltin diacetate. EiseiKagaku, 39(3) 219-225. [Pg.48]

Toop, J. and Emery, A. E. H. "Muscle Histology In Fetuses at Risk for Duchenne Muscular Dystrophy". Clin. Genet., (1974), 5, 230-233. [Pg.92]

Distribution studies by whole-body autoradiography in rats and monkeys revealed accumulation of radiolabel in the liver, kidney, lung, adrenal cortex and stomach. In fetuses exposed in utero, only the eye lens accumulated radiolabel at a higher concentration than that observed in maternal blood (Sandberg and Slanina 1980). [Pg.52]

Nickel sulfate 12-16 mg/kg BW on day 8 of gestation Reduction in mean number of live pups reduced body weight in fetuses on day 20 of gestation and in pups 4-8 weeks after birth 8... [Pg.507]

Hassoun, EA Creighton University Assess abilities of endrin to induce the formation of reactive oxygen species and lipid peroxidation and DNA single strand breaks that result in oxidative stress in fetuses of pregnant mice. The protective effects of some antioxidants will also be assessed using this model. [Pg.98]

No acute-duration inhalation MRL could be derived for mirex because no inhalation data could be located. No acute-duration oral MRL was derived for mirex because serious effects (heart block and arrhythmias in fetuses from dams exposed during gestation) were observed at the lowest dose tested (Grabowski 1983a). Studies examining the effects of mirex and chlordecone after acute-duration dermal exposure would be helpful since persons at hazardous waste sites may be exposed dermally to mirex. Additional dermal studies are certainly necessary because skin absorption of chlordecone appears to be an important route of exposure (Taylor et al. 1978). However, populations at hazardous waste sites are unlikely to be exposed via inhalation since these substances are virtually nonvolatile, so future studies using this route of exposure are not essential. [Pg.154]

Developmental Toxicity. No studies were located regarding developmental effects in humans after inhalation, oral, or dermal exposure to disulfoton or in animals after inhalation or dermal exposure. Developmental effects have been found in animals after acute- and intermediate-duration oral exposure to disulfoton. Plasma and erythrocyte cholinesterase depression and increased incidences of incomplete ossified parietal bones and sternebrae were observed in fetuses from rats fed... [Pg.133]

Carrier of cholesterol and phospholipids (HDL) Protease inhibitor iosmotic regulator in fetus Unknown... [Pg.176]

Saunders NR, Dziegielewska KM, Mollgard K, Risau W, Wolburg H (1991) The importance of the blood-brain barrier in fetuses and embryos. Trends Neurosci 14 14-15... [Pg.411]

Risk factors for deficiency Pregnancy (neural tube defects in fetus may result) Alcoholism Severe malnutrition Risk fiictors for deficiency Pernicious anemia Gastric resection Chronic pancreatitis Severe malnutrition Vegan Infection with D. latum... [Pg.251]

Ellis WG, Semple JL, Hoogenboom JR, Kavlock RJ, Zeman FJ Benomyl-induced craniocerebral anomalies in fetuses of adequately nourished and protein-deprived rats. Teratog Carcinog Mutagen 8 377-391, 1988... [Pg.68]

Daily oral administration of 600 or 800 mg/kg ronnel to dams on days 6 through 15 of gestation caused a significant dose-related increase in fetuses with an extra rib. ... [Pg.620]

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See also in sourсe #XX -- [ Pg.4 , Pg.153 ]



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