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Glutamate, decarboxylase receptor

GLP-1, glucagon like peptide-1 Glu, glutamate, glutamic acid GluDG, glutamate decarboxylase Glu-R, glutamate ionotropic receptor Gly, glycine... [Pg.842]

Mukuta T, Yoshikawa N, Arreaza G, et al. Activation of T lymphocyte subsets by synthetic TSH receptor peptides and recombinant glutamate decarboxylase in autoimmune thyroid disease and insulin-dependent diabetes. J CUn Endocrinol Metab 1995 80 1264-1272. [Pg.1389]

Increases occur in the activity and amount of glutamic acid decarboxylase. Glutamate levels are not affected by bilobalide, nor are the number or dissociation constants of the GABAA receptor. Such effects were suggested to mediate the antiseizure effects of bilobalide. [Pg.163]

Lombardi G., Szekely A. M., Bristol L. A., Guidotti A., and Manev H. (1993). Induction of ornithine decarboxylase by N-methyl-D-aspartate receptor activation is unrelated to potentiation of glutamate excitotoxicity by polyamines in cerebellar granule neurons. J. Neurochem. 60 1317-1324. [Pg.132]

Woo TU, Walsh JP, Benes FM. 2004. Density of glutamic acid decarboxylase 67 messenger RNA-containing neurons that express the N-methyl-D-aspartate receptor subunit NR2A in the anterior cingulate cortex in schizophrenia and bipolar disorder. Arch Gen Psychiatry 61 649-657. [Pg.491]


See other pages where Glutamate, decarboxylase receptor is mentioned: [Pg.175]    [Pg.326]    [Pg.1864]    [Pg.311]    [Pg.23]    [Pg.102]    [Pg.270]    [Pg.402]    [Pg.375]    [Pg.140]    [Pg.232]    [Pg.764]    [Pg.516]    [Pg.1120]    [Pg.246]    [Pg.336]    [Pg.67]    [Pg.430]    [Pg.292]    [Pg.84]    [Pg.54]    [Pg.285]    [Pg.235]    [Pg.507]    [Pg.121]    [Pg.524]    [Pg.615]    [Pg.375]    [Pg.21]    [Pg.114]    [Pg.114]    [Pg.602]    [Pg.569]    [Pg.651]    [Pg.78]    [Pg.263]    [Pg.288]    [Pg.40]    [Pg.384]    [Pg.424]   
See also in sourсe #XX -- [ Pg.164 , Pg.311 ]

See also in sourсe #XX -- [ Pg.164 , Pg.311 ]

See also in sourсe #XX -- [ Pg.164 , Pg.311 ]




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Glutamic decarboxylase

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