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Future for Cancer Epidemiology

An increased awareness of the health effects of chemical exposures has led to a reduction of chemical exposures in the workplace and the environment While there are undoubtedly substances that are yet to be causally associated with cancCT, the number of substances like those described in Table 15.6 is unlikely to grow significantly in the next decade. So where does the future for cancer epidemiology Ue  [Pg.414]

ACS (2007). Nutrition and Cancer. American Cancer Society. Available at http //www.cancer.org/ docroot/PRO/content/PRO l lx Nutrition.pdf.asp sitearea=PRO. Accessed January 19, 2009. [Pg.416]

and van Raaij, M. T. M. (2004). Risk assessment of peak exposure to genotoxic carcinogens A pragmatic approach. Toxicol Lett 151 (1), 43-50. [Pg.416]

and Chu, K. C. (1987). Additive and multiplicative models and multistage caroano enesis theory. Risk Anal 9 (1), 99-105. [Pg.416]

Castro, D. J., Lohr, C. V., Fischer, K. A., Perera, C. B., and Williams, D. E. (2008). Lymphoma and lung cancer in offspring born to pregnant mice dosed with dibenzo[a,/lpyrene The importance of in utero vs. lactational exposure. Toxicol Appl Pharmacol 233 (3), 454—458. [Pg.416]


See other pages where Future for Cancer Epidemiology is mentioned: [Pg.398]    [Pg.414]    [Pg.415]   


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