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Faah gene

Cravatt et al. (2001) generated mutant mice lacking the faah gene (FAAH / mice) and characterized by altered nociceptive threshold, enhanced memory extinction, and increased sensitivity to the effects of exogenously administered ECs (Cravatt et al., 2001 Varvel et al., 2007). Such behavioral phenotype is compatible with the higher AEA levels measured in these mice (Cravatt et al., 2001). [Pg.65]

Finally, a potential problem in human is the finding of a second faah gene, which is not present in rodents, although URB597 appears to be effective in inhibiting also this human FAAH isoform (Wei et al, 2006). [Pg.66]

Filbey FM, Schacht JP, Myers US (2010) Individual and additive effects of the CNR1 and FAAH genes on brain response to marijuana cues. Neuropsychopharmacology 35 967-975... [Pg.236]

Haughey HM, Marshall E, Schacht JP (2008) Marijuana withdrawal and craving influence of the cannabinoid receptor 1 (CNR1) and fatty acid amide hydrolase (FAAH) genes. Addiction 103 1678-1686... [Pg.236]

Mitjans M, Gasto C, Catalan R et al (2012) Genetic variability in the endocannabinoid system and 12-week clinical response to cital-opram treatment the role of the CNR1, CNR2 and FAAH genes. J Psychopharmacol 26 1391-1398... [Pg.237]

The genomic organization of mouse and human FAAH genes was reported in 1998 (Wan et ah, 1998). In humans, the gene is localized to chromosome... [Pg.118]

See other pages where Faah gene is mentioned: [Pg.466]    [Pg.60]    [Pg.466]    [Pg.10]    [Pg.11]    [Pg.159]    [Pg.190]    [Pg.287]    [Pg.287]    [Pg.566]    [Pg.101]    [Pg.118]    [Pg.118]    [Pg.119]    [Pg.82]    [Pg.87]    [Pg.101]    [Pg.262]    [Pg.543]   
See also in sourсe #XX -- [ Pg.60 , Pg.65 ]



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