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Dysimmune inflammatory polyneuropathy

The earliest reports of neurological complications of AIDS described distal symmetrical, painful sensory neuropathy occurring in HIV patients (Snider et al. 1983). Dysimmune inflammatory polyneuropathy was subsequently recognized as a complication of AIDS (Lipkin et al. 1985). Progressive polyneuropathy associated with cytomegalovirus (CMV) infection was documented as the first truly opportunistic infection of the peripheral nerve (Eidelberg et al. 1986). [Pg.52]

In a patient who does have a close family history of diabetes-2, a glucocorticoid should be avoided if possible, because it can increase insulin resistance and make manifest diabetes-2 glucose dysmetabo-lism. Accordingly, asking about a close family history of diabetes-2 is very important in managing a chronic immune dysschwannian polyneuropathy (CIDP) (sometimes less precisely called chronic inflammatory demyelinating polyneuropathy), or other dysimmune patient. [Pg.36]

Treatment Because of the sensory abnormalities, dysschwannian nerve conductions, elevated CSF protein, aspects of denervation-reinnervation in the muscle biopsy, and the diabetes-2 in both parents, we considered that our patient probably had coexisting genetico-diahetoid-2 dysimmune neuropathy," a type of chronic immune dysschwannian polyneuropathy (CIDP) (sometimes less precisely called chronic inflammatory demyelinating polyneuropathy, Chapter 2). We therefore treated her using/WG, and she was remarkably benefited her walking ability and endurance greatly improved, she did not require frequent rests, and was able to ascend steps much more easily. [Pg.61]


See also in sourсe #XX -- [ Pg.52 ]




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