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D-amino levulinate

Hodson, P.V. 1976. d-amino levulinic acid dehydratase activity of fish blood as an indicator of a harmful exposure to lead. Jour. Fish. Res. Board Can. 33 268-271. [Pg.333]

Mauzerall D, Granick S (1995) The occurrence and determination of delta-amino-levulinic acid and porphobilinogen in urine. J Biol Chem 219 435-446... [Pg.780]

At least three polymorphic genes have been identified that potentially can influence the bioaccumulation and toxicokinetics of lead in humans (1) the gene coding for 6-amino-levulinic acid dehydratase (ALAD) (2) the Vitamin D receptor (VDR) gene and (3) HFE, the gene for hereditary hemochromatosis (Onalaja et al. 2000). [Pg.469]

Louis, E.D., L. Applegate, J.H. Graziano, M. Parides, V. Slavkovich, and H.K. Bhat. 2005. Interaction between blood lead concentration and delta-amino-levulinic acid dehydratase gene polymorphisms increases the odds of essential tremor. Mov. Disord. 20(9) 1170-1177. [Pg.138]

Tschudy, D. P., Marver, H. S., and Collins, A., 1965, A model for calculating messenger RNA half life Short lived messenger RNA in the induction of mammalian S-amino-levulinic acid synthetase, Biochem. Biophys. Res. Cotnmun. 21 480. [Pg.264]

See other pages where D-amino levulinate is mentioned: [Pg.573]    [Pg.29]    [Pg.573]    [Pg.29]    [Pg.129]    [Pg.561]    [Pg.568]    [Pg.175]    [Pg.234]    [Pg.258]    [Pg.78]    [Pg.679]   
See also in sourсe #XX -- [ Pg.454 ]



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Levulinate

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