Corticosteroids hypokalemia with

Oral Treatment of hypokalemia in the following conditions With or without metabolic alkalosis digitalis intoxication familial periodic paralysis diabetic acidosis diarrhea and vomiting surgical conditions accompanied by nitrogen loss, vomiting, suction drainage, diarrhea, and increased urinary excretion of potassium certain cases of uremia hyperadrenalism starvation and debilitation corticosteroid or diuretic therapy. 

Fluid/Solute overload Excessive amounts of sodium chloride by any route may cause hypokalemia and acidosis. Administration of IV solutions can cause fluid or solute overload resulting in dilution of serum electrolyte concentrations, CHF, overhydration, congested states, or acute pulmonary edema, especially in patients with cardiovascular disease and in patients receiving corticosteroids or corticotropin or drugs that may give rise to sodium retention. 

Hypokalemia - Hypokalemia may develop during concomitant corticosteroids, ACTH, and especially with brisk diuresis, with severe liver disease or cirrhosis, vomiting or diarrhea, or after prolonged therapy. 

Thiazide drugs have additive potassium-lowering effects with other drugs that reduce serum potassium, such as corticosteroids and carbenoxolone. Profound hypokalemia due to the latter combination has been reported to cause massive rhabdomyolysis and acute tubular necrosis (49). 

With long-term use hypokalemia due to intestinal potassium loss could occur (Blumenthal, 1998). Thiazide diuretics, corticosteroids, and licorice could exacerbate hypokalemia. The effects of digoxin can be potentiated by hypokalemia. 

Corticosteroids Digitalis glycosides CO-administration may enhance the possibility of digitalis toxicity assodated with hypokalemia. 

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