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Axons dying-back" degeneration

Histological examination of hens exposed to TOCP revealed a wallerian dying-back degeneration of the larger diameter axons and myelin sheaths. If the neuropathic target esterase is inhibited by 70%, the typical organophophorus ester-induced delayed neurotoxicity (OPIDN) will follow after an approximate 7-14 day delay. [Pg.1253]

Spencer PS, Schaumburg HH. 1977a. Ultrastructural studies of the dying-back process. III. The evolution of experimental peripheral giant axonal degeneration. J Neuropathol Exp Neurol 36(2) 276-299. [Pg.246]

A number of pathological changes are seen in the CNS of ALS patients, with an expected regional distribution. The most prominent of these is a loss of large motoneurons in the motor cortex, brain stem and spinal cord. It has been debated whether this degeneration represents a dying back mechanism, spreading from the axon back to the... [Pg.431]

See other pages where Axons dying-back" degeneration is mentioned: [Pg.737]    [Pg.658]    [Pg.746]    [Pg.56]    [Pg.66]    [Pg.62]    [Pg.732]    [Pg.67]    [Pg.329]    [Pg.42]    [Pg.348]    [Pg.288]    [Pg.733]    [Pg.733]    [Pg.733]    [Pg.734]    [Pg.1886]    [Pg.66]    [Pg.67]    [Pg.573]    [Pg.286]    [Pg.77]    [Pg.252]    [Pg.27]   
See also in sourсe #XX -- [ Pg.746 ]



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