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Trichlorfon neurotoxicity

A number of OPC are capable of rendering a delayed neurotoxic effect (DNE). This effect becomes apparent gradually, after a certain latent period (usually 14 to 21 days, sometimes 1 to 5 years after the acute poisoning survived) and is characterized clinically by the development of ataxia, muscular weakness, paresis and paralysis of the extremities. Morphologically, it is characterized by fiber demyelinization of spinal pathways and peripheral nerves. Till present time, near 40,000 cases have been described, when paresis and paralysis developed in human beings as a result of their exposure to OPC (TOCP, mipaphox, chloropyrophos, trichlorfon, etc.) [1],... [Pg.103]

See other pages where Trichlorfon neurotoxicity is mentioned: [Pg.1219]    [Pg.60]    [Pg.1372]    [Pg.384]    [Pg.2769]    [Pg.25]    [Pg.110]    [Pg.173]    [Pg.340]    [Pg.141]    [Pg.111]   
See also in sourсe #XX -- [ Pg.24 ]



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Trichlorfon

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