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Potassium channels synaptic plasticity

HT4 Receptor Regulation of Voltage-Dependent Potassium Channels Possible Roles in the Regulation of Neurotransmitter Release and Synaptic Plasticity... [Pg.485]

Fig. 2 Substance P induced NTS plasticity targets in the cough reflex. Plasticity targets include (1) increased intrinsic excitabUity via closure of potassium channels, activation of nonselective cation channels, opening of L-type calcium channel, and intracellular calcium release. (2) increased synaptic excitability via an increased ionotropic glutamate receptor function, a decreased GABAa receptor function. Other compensatory mechanisms may also occur, including an increased GABA release and decreased glutamate release from the afferent terminals. AP area postrema... Fig. 2 Substance P induced NTS plasticity targets in the cough reflex. Plasticity targets include (1) increased intrinsic excitabUity via closure of potassium channels, activation of nonselective cation channels, opening of L-type calcium channel, and intracellular calcium release. (2) increased synaptic excitability via an increased ionotropic glutamate receptor function, a decreased GABAa receptor function. Other compensatory mechanisms may also occur, including an increased GABA release and decreased glutamate release from the afferent terminals. AP area postrema...
See other pages where Potassium channels synaptic plasticity is mentioned: [Pg.99]    [Pg.55]    [Pg.514]    [Pg.252]    [Pg.436]    [Pg.212]    [Pg.157]    [Pg.157]   
See also in sourсe #XX -- [ Pg.485 , Pg.486 ]



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