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Platelet-activating factor catabolism

Hyperglycemia followed by hypoglycemia Catabolic hormones release Platelet-activating factor and eicosanoid release... [Pg.319]

Snyder F. (1995). Platelet-activating factor the biosynthetic and catabolic enzymes. Biochem. J. 305 689-705. [Pg.102]

Fig. 13. Catabolism of platelet-activating factor (PAF) and its metabolites by (I) PAF-AH, (II) lysophospholipase D, (III) phosphohydrolase, (IV) phospholipase C, (V) Cok-independent or CoA-dependenI transacylase, and/or (VI) alkylacetylglycerol acetylhydrolase. The O-alkyl linkage in products that contain free hydroxyl groups can be cleaved by (VII) the O-alkyl Pte H4-dependent monooxygenase. Fig. 13. Catabolism of platelet-activating factor (PAF) and its metabolites by (I) PAF-AH, (II) lysophospholipase D, (III) phosphohydrolase, (IV) phospholipase C, (V) Cok-independent or CoA-dependenI transacylase, and/or (VI) alkylacetylglycerol acetylhydrolase. The O-alkyl linkage in products that contain free hydroxyl groups can be cleaved by (VII) the O-alkyl Pte H4-dependent monooxygenase.
Activated platelet-derived factors must undergo normal catabolism. Factors that are already activated do not depend on the presence of vitamin K, so the effect of the drug is only seen after normal catabolism of activated factors. [Pg.154]

See other pages where Platelet-activating factor catabolism is mentioned: [Pg.345]    [Pg.495]    [Pg.22]    [Pg.104]    [Pg.124]    [Pg.247]    [Pg.247]    [Pg.25]   
See also in sourсe #XX -- [ Pg.269 ]



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