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Peptidase pulmonary

Lilly, C.M., Martins, M.A. and Drazen, J.M. (1993a). Peptidase modulation of vasoactive intestinal peptide pulmonary relaxation in tracheal superfused guinea-pig lung . J. Clin. Invest. 91, 235-243. [Pg.142]

Many authors have investigated the influence of ozone on pulmonary lysosomes and mitochondria. Lysosomes contain quite many enzymes, mainly hydrolases, such as protease, lipases, nucleases, phosphatases, phosphodiesterases, etc. Dillar et al. [273] have studied the changes in lysosome levels upon exposure of mice to ozone (0.7 ppm). The activity of pulmonary lysosomal cathepsin A, cathepsin D, acidic phosphatase, (3-N-acetylglucose aminidase, and benzyl arginine P-naphthylamide ami-dohydrolase was increased. This is due to inflammatory processes caused by ozone. The specific activity of protease and peptidase in the lung is enhanced when related to chronic obstructive genetic lack of a-1-antitrypsin factor. [Pg.167]

See other pages where Peptidase pulmonary is mentioned: [Pg.361]    [Pg.130]    [Pg.274]    [Pg.316]    [Pg.222]    [Pg.138]    [Pg.36]    [Pg.76]    [Pg.112]    [Pg.113]    [Pg.144]    [Pg.351]    [Pg.816]    [Pg.1463]    [Pg.400]    [Pg.223]    [Pg.305]    [Pg.217]    [Pg.340]    [Pg.229]   
See also in sourсe #XX -- [ Pg.286 ]



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